Debashree Mukherjea

Expression and function of placenta growth factor: implications for abnormal placentation.

Essential role of Rac1/NADPH oxidase in nerve growth factor induction of TRPV1 expression.

Induction of adenosine A1 receptor expression by pertussis toxin via an adenosine 5'-diphosphate ribosylation-independent pathway.

Activation of the adenosine A1 receptor inhibits HIV-1 tat-induced apoptosis by reducing nuclear factor-kappaB activation and inducible nitric-oxide synthase.

Glial cell missing 1 regulates placental growth factor (PGF) gene transcription in human trophoblast.

Short interfering RNA against transient receptor potential vanilloid 1 attenuates cisplatin-induced hearing loss in the rat.

Role of beta-arrestin1/ERK MAP kinase pathway in regulating adenosine A1 receptor desensitization and recovery.

Cisplatin ototoxicity and protection: clinical and experimental studies.

Adenosine A(3) receptor suppresses prostate cancer metastasis by inhibiting NADPH oxidase activity.

Transtympanic administration of short interfering (si)RNA for the NOX3 isoform of NADPH oxidase protects against cisplatin-induced hearing loss in the rat.

Pharmacogenomics of cisplatin-induced ototoxicity.

The design and screening of drugs to prevent acquired sensorineural hearing loss.

Resveratrol reduces prostate cancer growth and metastasis by inhibiting the Akt/MicroRNA-21 pathway.

Essential Role of NADPH Oxidase-Dependent Reactive Oxygen Species Generation in Regulating MicroRNA-21 Expression and Function in Prostate Cancer.

Adenosine receptors: expression, function and regulation.

TRPV1: A Potential Drug Target for Treating Various Diseases.

Early investigational drugs for hearing loss.

Adenosine A1 Receptor Protects Against Cisplatin Ototoxicity by Suppressing the NOX3/STAT1 Inflammatory Pathway in the Cochlea.

Tonic suppression of PCAT29 by the IL-6 signaling pathway in prostate cancer: Reversal by resveratrol.

Epigallocatechin-3-gallate, a prototypic chemopreventative agent for protection against cisplatin-based ototoxicity.

Mechanisms of Cisplatin-Induced Ototoxicity and Otoprotection.