Alessandro Fantin

Tissue macrophages act as cellular chaperones for vascular anastomosis downstream of VEGF-mediated endothelial tip cell induction.

The cytoplasmic domain of neuropilin 1 is dispensable for angiogenesis, but promotes the spatial separation of retinal arteries and veins.

NRP1 acts cell autonomously in endothelium to promote tip cell function during sprouting angiogenesis.

The embryonic mouse hindbrain as a qualitative and quantitative model for studying the molecular and cellular mechanisms of angiogenesis.

The neuropilin 1 cytoplasmic domain is required for VEGF-A-dependent arteriogenesis.

The cytoplasmic domain of neuropilin-1 regulates focal adhesion turnover.

Neuropilin 1 (NRP1) hypomorphism combined with defective VEGF-A binding reveals novel roles for NRP1 in developmental and pathological angiogenesis.

Neuropilin regulation of angiogenesis, arteriogenesis and vascular permeability.

Imatinib inhibits VEGF-independent angiogenesis by targeting neuropilin 1-dependent ABL1 activation in endothelial cells.

The mouse hindbrain: an in vivo model to analyze developmental angiogenesis.

VEGF189 binds NRP1 and is sufficient for VEGF/NRP1-dependent neuronal patterning in the developing brain.

Neural crest cells in cardiovascular development.

Dysfunctional SEMA3E signaling underlies gonadotropin-releasing hormone neuron deficiency in Kallmann syndrome.

NRP1 Regulates CDC42 Activation to Promote Filopodia Formation in Endothelial Tip Cells.

Neural crest-derived SEMA3C activates endothelial NRP1 for cardiac outflow tract septation.

Vascularisation of the central nervous system.

The Embryonic Mouse Hindbrain and Postnatal Retina as In Vivo Models to Study Angiogenesis.

Myeloid-Derived Vascular Endothelial Growth Factor and Hypoxia-Inducible Factor Are Dispensable for Ocular Neovascularization--Brief Report.

NRP1 function and targeting in neurovascular development and eye disease.

2- and 6-O-sulfated proteoglycans have distinct and complementary roles in cranial axon guidance and motor neuron migration.

Neuropilin-1 mediates vascular permeability independently of vascular endothelial growth factor receptor-2 activation.

A Small Molecule Inhibitor of PDK1/PLCγ1 Interaction Blocks Breast and Melanoma Cancer Cell Invasion.

Imatinib may be ABL to improve anti-angiogenic therapy.

VEGF165-induced vascular permeability requires NRP1 for ABL-mediated SRC family kinase activation.