Gregory L. Brower

Estrogenic modulation of inflammation-related genes in male rats following volume overload.

Effects of mast cells on the behavior of isolated heart fibroblasts: modulation of collagen remodeling and gene expression.

Effects of matrix metalloproteinase inhibition on ventricular remodeling due to volume overload.

Cardiac mast cell-mediated activation of gelatinase and alteration of ventricular diastolic function.

Gender differences in cardiac remodeling secondary to chronic volume overload.

Cause and effect relationship between myocardial mast cell number and matrix metalloproteinase activity.

The role of myocardial fibrillar collagen in ventricular remodeling and function.

Cardiac mast cell- and chymase-mediated matrix metalloproteinase activity and left ventricular remodeling in mitral regurgitation in the dog.

Gender mediated cardiac protection from adverse ventricular remodeling is abolished by ovariectomy.

The dynamic interaction between matrix metalloproteinase activity and adverse myocardial remodeling.

Endothelin-1 mediates cardiac mast cell degranulation, matrix metalloproteinase activation, and myocardial remodeling in rats.

Modulation of cardiac mast cell-mediated extracellular matrix degradation by estrogen.

Effects of dietary phytoestrogens on cardiac remodeling secondary to chronic volume overload in female rats.

Pharmacologic inhibition of mast cell degranulation prevents left ventricular remodeling induced by chronic volume overload in rats.

Cardiac mast cell regulation of matrix metalloproteinase-related ventricular remodeling in chronic pressure or volume overload.

The relationship between myocardial extracellular matrix remodeling and ventricular function.

Inhibition of matrix metalloproteinase activity by ACE inhibitors prevents left ventricular remodeling in a rat model of heart failure.

Response of cardiac mast cells to atrial natriuretic peptide.

Cardioprotection in female rats subjected to chronic volume overload: synergistic interaction of estrogen and phytoestrogens.

Effects of nonselective endothelin-1 receptor antagonism on cardiac mast cell-mediated ventricular remodeling in rats.

Defective intracellular Ca2+ homeostasis contributes to myocyte dysfunction during ventricular remodelling induced by chronic volume overload in rats.

Protection from adverse myocardial remodeling secondary to chronic volume overload in mast cell deficient rats.

Regulation of matrix metalloproteinases is at the heart of myocardial remodeling.

Cardiac mast cells mediate left ventricular fibrosis in the hypertensive rat heart.

ETA selective receptor antagonism prevents ventricular remodeling in volume-overloaded rats.

Nrf2 protects against maladaptive cardiac responses to hemodynamic stress.

TNF-alpha inhibition attenuates adverse myocardial remodeling in a rat model of volume overload.

Estrogen attenuates chronic volume overload induced structural and functional remodeling in male rat hearts.

Sympathetic nervous system modulation of inflammation and remodeling in the hypertensive heart.

Inhibition of matrix metalloproteinase activity prevents increases in myocardial tumor necrosis factor-alpha.

Interleukin 6 mediates myocardial fibrosis, concentric hypertrophy, and diastolic dysfunction in rats.

Temporal evaluation of cardiac myocyte hypertrophy and hyperplasia in male rats secondary to chronic volume overload.

Cardiac mast cells: the centrepiece in adverse myocardial remodelling.

Tryptase/Protease-activated receptor 2 interactions induce selective mitogen-activated protein kinase signaling and collagen synthesis by cardiac fibroblasts.