Adam Bailis

Multiple pathways promote short-sequence recombination in Saccharomyces cerevisiae.

Nucleotide Excision Repair, Genome Stability, and Human Disease: New Insight from Model Systems.

DNA fragment transplacement in Saccharomyces cerevisiae: some genetic considerations.

Telomere dysfunction drives increased mutation by error-prone polymerases Rev1 and zeta in Saccharomyces cerevisiae.

A mutant allele of the transcription factor IIH helicase gene, RAD3, promotes loss of heterozygosity in response to a DNA replication defect in Saccharomyces cerevisiae.

RAD59 is required for efficient repair of simultaneous double-strand breaks resulting in translocations in Saccharomyces cerevisiae.

Mating type influences chromosome loss and replicative senescence in telomerase-deficient budding yeast by Dnl4-dependent telomere fusion.

Mutagenic and recombinagenic responses to defective DNA polymerase delta are facilitated by the Rev1 protein in pol3-t mutants of Saccharomyces cerevisiae.

Telomerase deficiency affects the formation of chromosomal translocations by homologous recombination in Saccharomyces cerevisiae.

Msh2 blocks an alternative mechanism for non-homologous tail removal during single-strand annealing in Saccharomyces cerevisiae.

RAD59 and RAD1 cooperate in translocation formation by single-strand annealing in Saccharomyces cerevisiae.

Rad51 inhibits translocation formation by non-conservative homologous recombination in Saccharomyces cerevisiae.

RAD51C germline mutations in breast and ovarian cancer cases from high-risk families.