Peer Wulff

Impaired renal Na(+) retention in the sgk1-knockout mouse.

Impaired regulation of renal K+ elimination in the sgk1-knockout mouse.

Affinity of various benzodiazepine site ligands in mice with a point mutation in the GABA(A) receptor gamma2 subunit.

Exploitation of KESTREL to identify NDRG family members as physiological substrates for SGK1 and GSK3.

Role of Sgk1 in salt and potassium homeostasis.

Deranged Kv channel regulation in fibroblasts from mice lacking the serum and glucocorticoid inducible kinase SGK1.

Dissecting neural circuitry by combining genetics and pharmacology.

Regulation of the excitatory amino acid transporter EAAT5 by the serum and glucocorticoid dependent kinases SGK1 and SGK3.

Serum- and glucocorticoid-inducible kinase 1 (SGK1) mediates glucocorticoid-induced inhibition of insulin secretion.

SGK1 as a determinant of kidney function and salt intake in response to mineralocorticoid excess.

Blunted hypertensive effect of combined fructose and high-salt diet in gene-targeted mice lacking functional serum- and glucocorticoid-inducible kinase SGK1.

Intestinal function of gene-targeted mice lacking serum- and glucocorticoid-inducible kinase 1.

Renal function of gene-targeted mice lacking both SGK1 and SGK3.

SGK1-dependent cardiac CTGF formation and fibrosis following DOCA treatment.

Renal Ca2+ handling in sgk1 knockout mice.

Serum- and glucocorticoid-inducible kinase 1 mediates salt sensitivity of glucose tolerance.

Blunted DOCA/high salt induced albuminuria and renal tubulointerstitial damage in gene-targeted mice lacking SGK1.

Resistance of mice lacking the serum- and glucocorticoid-inducible kinase SGK1 against salt-sensitive hypertension induced by a high-fat diet.

From synapse to behavior: rapid modulation of defined neuronal types with engineered GABAA receptors.

Role of the serum and glucocorticoid inducible kinase SGK1 in glucocorticoid stimulation of gastric acid secretion.

Hippocampal theta rhythm and its coupling with gamma oscillations require fast inhibition onto parvalbumin-positive interneurons.

Synaptic inhibition of Purkinje cells mediates consolidation of vestibulo-cerebellar motor learning.

Studying Cerebellar Circuits by Remote Control of Selected Neuronal Types with GABA(A) Receptors.

Quantitative organization of GABAergic synapses in the molecular layer of the mouse cerebellar cortex.

Ro 15-4513 Antagonizes Alcohol-Induced Sedation in Mice Through αβγ2-type GABA(A) Receptors.

Parvalbumin-positive CA1 interneurons are required for spatial working but not for reference memory.

Actions of two GABAA receptor benzodiazepine-site ligands that are mediated via non-γ2-dependent modulation.

Removal of GABA(A) receptor γ2 subunits from parvalbumin neurons causes wide-ranging behavioral alterations.

Chemical genetics: receptor-ligand pairs for rapid manipulation of neuronal activity.

The hippocampus in aging and disease: From plasticity to vulnerability.

Parvalbumin-positive interneurons of the prefrontal cortex support working memory and cognitive flexibility.

Parvalbumin-containing GABA cells and schizophrenia: experimental model based on targeted gene delivery through adeno-associated viruses.

Spiny and Non-spiny Parvalbumin-Positive Hippocampal Interneurons Show Different Plastic Properties.

Afferent connections of the thalamic nucleus reuniens in the mouse.

Cerebellar molecular layer interneurons are dispensable for cued and contextual fear conditioning.