Jonathan Ewbank

Tackling both sides of the host-pathogen equation with Caenorhabditis elegans.

Diverse bacteria are pathogens of Caenorhabditis elegans.

Inducible antibacterial defense system in C. elegans.

Virulence factors of the human opportunistic pathogen Serratia marcescens identified by in vivo screening.

Caenorhabditis elegans: an emerging genetic model for the study of innate immunity.

Q & A. Jonathan Ewbank.

[A worm's life].

Immunity in Caenorhabditis elegans.

[The nematode Caenorhabditis elegans as a model for the study of host-pathogen interactions].

TLR-independent control of innate immunity in Caenorhabditis elegans by the TIR domain adaptor protein TIR-1, an ortholog of human SARM.

Evolution of the innate immune system: the worm perspective.

Diversity and specificity in the interaction between Caenorhabditis elegans and the pathogen Serratia marcescens.

Genetic models in pathogenesis.

XNP-1/ATR-X acts with RB, HP1 and the NuRD complex during larval development in C. elegans.

'TaiLoRing' the response of dendritic cells to pathogens.

A generalized transducing phage (phiIF3) for the genomically sequenced Serratia marcescens strain Db11: a tool for functional genomics of an opportunistic human pathogen.

A semi-automated high-throughput approach to the generation of transposon insertion mutants in the nematode Caenorhabditis elegans.

Infection in a dish: high-throughput analyses of bacterial pathogenesis.

Detection and avoidance of a natural product from the pathogenic bacterium Serratia marcescens by Caenorhabditis elegans.

Genome-wide investigation reveals pathogen-specific and shared signatures in the response of Caenorhabditis elegans to infection.

The genetics of pathogen avoidance in Caenorhabditis elegans.

A model of bacterial intestinal infections in Drosophila melanogaster.

Signaling in the immune response.

Pathogenomics: an updated European Research Agenda.

Distinct innate immune responses to infection and wounding in the C. elegans epidermis.

Anti-fungal innate immunity in C. elegans is enhanced by evolutionary diversification of antimicrobial peptides.

Genome sequence of the metazoan plant-parasitic nematode Meloidogyne incognita.

Negative regulation of Caenorhabditis elegans epidermal damage responses by death-associated protein kinase.

Neuroimmune regulation of antimicrobial peptide expression by a noncanonical TGF-beta signaling pathway in Caenorhabditis elegans epidermis.

Antifungal innate immunity in C. elegans: PKCdelta links G protein signaling and a conserved p38 MAPK cascade.

Caenorhabditis elegans semi-automated liquid screen reveals a specialized role for the chemotaxis gene cheB2 in Pseudomonas aeruginosa virulence.

SMF-1, SMF-2 and SMF-3 DMT1 orthologues regulate and are regulated differentially by manganese levels in C. elegans.

Cellular homeostasis: coping with ER overload during an immune response.

The fatty acid synthase fasn-1 acts upstream of WNK and Ste20/GCK-VI kinases to modulate antimicrobial peptide expression in C. elegans epidermis.

C. elegans: model host and tool for antimicrobial drug discovery.

Unusual regulation of a STAT protein by an SLC6 family transporter in C. elegans epidermal innate immunity.

A comprehensive analysis of gene expression changes provoked by bacterial and fungal infection in C. elegans.

A Genome-Wide Collection of Mos1 Transposon Insertion Mutants for the C. elegans Research Community.