Antonina Joanna Mazur

Gelsolin affects the migratory ability of human colon adenocarcinoma and melanoma cells.

Cofilin overexpression affects actin cytoskeleton organization and migration of human colon adenocarcinoma cells.

Effect of overexpression of β- and γ-actin isoforms on actin cytoskeleton organization and migration of human colon cancer cells.

Analysis of gelsolin expression pattern in developing chicken embryo reveals high GSN expression level in tissues of neural crest origin.

Active invadopodia of mesenchymally migrating cancer cells contain both β and γ cytoplasmic actin isoforms.

Gelsolin interacts with LamR, hnRNP U, nestin, Arp3 and β-tubulin in human melanoma cells as revealed by immunoprecipitation and mass spectrometry.

Involvement of β- and γ-actin isoforms in actin cytoskeleton organization and migration abilities of bleb-forming human colon cancer cells.

Are non-muscle actin isoforms functionally equivalent?

Gefitinib or lapatinib with foretinib synergistically induce a cytotoxic effect in melanoma cell lines.

Thymosin β4 Regulates Focal Adhesion Formation in Human Melanoma Cells and Affects Their Migration and Invasion.

Knockout of and with CRISPR/Cas9(D10A) Technique Shows that Non-Muscle β and γ Actin Are Not Equal in Relation to Human Melanoma Cells' Motility and Focal Adhesion Formation.

The origin of the expressed retrotransposed gene ACTBL2 and its influence on human melanoma cells' motility and focal adhesion formation.

A Fluorescent Gelatin Degradation Assay to Study Melanoma Breakdown of Extracellular Matrix.

Changes in Biomechanical Properties of A375 Cells Due to the Silencing of Expression Are Not Directly Correlated with Alterations in Their Stemness Features.

Integrin-Linked Kinase (ILK) Plays an Important Role in the Laminin-Dependent Development of Dorsal Root Ganglia during Chicken Embryogenesis.

Gelsolin Contributes to the Motility of A375 Melanoma Cells and This Activity Is Mediated by the Fibrous Extracellular Matrix Protein Profile.

Integrin-linked kinase (ILK): the known vs. the unknown and perspectives.

SLC35A2 Deficiency Promotes an Epithelial-to-Mesenchymal Transition-like Phenotype in Madin-Darby Canine Kidney Cells.