Vickery Trinkaus-Randall

Wounding the cornea to learn how it heals.

P2Y receptors play a critical role in epithelial cell communication and migration.

Cellular response of cardiac fibroblasts to amyloidogenic light chains.

Modulation of endothelial cell migration by extracellular nucleotides: involvement of focal adhesion kinase and phosphatidylinositol 3-kinase-mediated pathways.

Role of endocytic inhibitory drugs on internalization of amyloidogenic light chains by cardiac fibroblasts.

Injury and nucleotides induce phosphorylation of epidermal growth factor receptor: MMP and HB-EGF dependent pathway.

Morphologic characterization of organized extracellular matrix deposition by ascorbic acid-stimulated human corneal fibroblasts.

Tri-nucleotide receptors play a critical role in epithelial cell wound repair.

Regulation by P2X7: epithelial migration and stromal organization in the cornea.

RelB NF-kappaB represses estrogen receptor alpha expression via induction of the zinc finger protein Blimp1.

The P2Y2 receptor mediates the epithelial injury response and cell migration.

Role of glycosaminoglycan sulfation in the formation of immunoglobulin light chain amyloid oligomers and fibrils.

Distinct activation of epidermal growth factor receptor by UTP contributes to epithelial cell wound repair.

Doxycycline reduces fibril formation in a transgenic mouse model of AL amyloidosis.

Corneal epithelium expresses a variant of P2X(7) receptor in health and disease.

The P2X(7) receptor regulates proteoglycan expression in the corneal stroma.

Disorganized collagen scaffold interferes with fibroblast mediated deposition of organized extracellular matrix in vitro.

Communication between corneal epithelial cells and trigeminal neurons is facilitated by purinergic (P2) and glutamatergic receptors.

New insights in wound response and repair of epithelium.

TGF-β3 stimulates stromal matrix assembly by human corneal keratocyte-like cells.

Epithelial wounds induce differential phosphorylation changes in response to purinergic and EGF receptor activation.

Hypoxia-induced changes in Ca(2+) mobilization and protein phosphorylation implicated in impaired wound healing.

Self-assembled matrix by umbilical cord stem cells.

Purines in the eye: recent evidence for the physiological and pathological role of purines in the RPE, retinal neurons, astrocytes, Müller cells, lens, trabecular meshwork, cornea and lacrimal gland.

Extracellular matrix stiffness modulates VEGF calcium signaling in endothelial cells: individual cell and population analysis.

Purinoreceptor P2X7 Regulation of Ca(2+) Mobilization and Cytoskeletal Rearrangement Is Required for Corneal Reepithelialization after Injury.

Purinergic Signaling in Corneal Wound Healing: A Tale of 2 Receptors.

Early life allergen-induced mucus overproduction requires augmented neural stimulation of pulmonary neuroendocrine cell secretion.

Hypoxia modulates the development of a corneal stromal matrix model.

High fat diet induces pre-type 2 diabetes with regional changes in corneal sensory nerves and altered P2X7 expression and localization.

Epithelial cells exert differential traction stress in response to substrate stiffness.

The Role of Hypoxia in Corneal Extracellular Matrix Deposition and Cell Motility.

Sustained Ca2+ mobilizations: A quantitative approach to predict their importance in cell-cell communication and wound healing.

Changes in Epithelial and Stromal Corneal Stiffness Occur with Age and Obesity.

Multiple Imaging Modalities for Cell-Cell Communication via Calcium Mobilizations in Corneal Epithelial Cells.

Pannexin1: Role as a Sensor to Injury Is Attenuated in Pretype 2 Corneal Diabetic Epithelium.

Age Dependent Changes in Corneal Epithelial Cell Signaling.