Alan S. Waldman

Multiple pathways for repair of DNA double-strand breaks in mammalian chromosomes.

Promiscuous patching of broken chromosomes in mammalian cells with extrachromosomal DNA.

Suppression of high-fidelity double-strand break repair in mammalian chromosomes by pifithrin-alpha, a chemical inhibitor of p53.

Determination of intrachromosomal recombination rates in cultured mammalian cells.

Modulation of error-prone double-strand break repair in mammalian chromosomes by DNA mismatch repair protein Mlh1.

A role for DNA mismatch repair protein Msh2 in error-prone double-strand-break repair in mammalian chromosomes.

Genetic exchange between homeologous sequences in mammalian chromosomes is averted by local homology requirements for initiation and resolution of recombination.

Accurate homologous recombination is a prominent double-strand break repair pathway in mammalian chromosomes and is modulated by mismatch repair protein Msh2.

Induction of intrachromosomal homologous recombination in human cells by raltitrexed, an inhibitor of thymidylate synthase.

DNA damage and homologous recombination signaling induced by thymidylate deprivation.

Ensuring the fidelity of recombination in mammalian chromosomes.

Wild-type p53-induced phosphatase 1 dephosphorylates histone variant gamma-H2AX and suppresses DNA double strand break repair.

Depletion of the bloom syndrome helicase stimulates homology-dependent repair at double-strand breaks in human chromosomes.

Expression and regulation of RAD51 mediate cellular responses to chemotherapeutics.

Induction of recombination between diverged sequences in a mammalian genome by a double-strand break.

Intrachromosomal recombination between highly diverged DNA sequences is enabled in human cells deficient in Bloom helicase.

High homology is not required at the site of strand invasion during recombinational double-strand break repair in mammalian chromosomes.

Mismatch tolerance during homologous recombination in mammalian cells.

Alteration of genetic recombination and double-strand break repair in human cells by progerin expression.

Corruption of DNA end-joining in mammalian chromosomes by progerin expression.

Progerin Can Induce DNA Damage in the Absence of Global Changes in Replication or Cell Proliferation.

Progerin can induce DNA damage in the absence of global changes in replication or cell proliferation.