Irena Pastar

Farnesyl pyrophosphate inhibits epithelialization and wound healing through the glucocorticoid receptor.

Attenuation of the transforming growth factor beta-signaling pathway in chronic venous ulcers.

Cortisol synthesis in epidermis is induced by IL-1 and tissue injury.

Keratin dressings speed epithelialization of deep partial-thickness wounds.

Micro-RNAs: New Regulators of Wound Healing.

Induction of specific microRNAs inhibits cutaneous wound healing.

Interactions of methicillin resistant Staphylococcus aureus USA300 and Pseudomonas aeruginosa in polymicrobial wound infection.

Quality assessment of tissue specimens for studies of diabetic foot ulcers.

Glucocorticoid receptor localizes to adherens junctions at the plasma membrane of keratinocytes.

Increased number of Langerhans cells in the epidermis of diabetic foot ulcers correlates with healing outcome.

Deregulation of epidermal stem cell niche contributes to pathogenesis of nonhealing venous ulcers.

Epithelialization in Wound Healing: A Comprehensive Review.

The Role of TGFβ Signaling in Wound Epithelialization.

Nanoparticles for Fidgety Cell Movement and Enhanced Wound Healing.

Comparative Genomic, MicroRNA, and Tissue Analyses Reveal Subtle Differences between Non-Diabetic and Diabetic Foot Skin.

Stem Cells in Skin Regeneration, Wound Healing, and Their Clinical Applications.

Identification of novel cyclic lipopeptides from a positional scanning combinatorial library with enhanced antibacterial and antibiofilm activities.

Skin Metabolite, Farnesyl Pyrophosphate, Regulates Epidermal Response to Inflammation, Oxidative Stress, and Migration.

Epithelial-mesenchymal transition in tissue repair and fibrosis.

Biology and Biomarkers for Wound Healing.

Integrative analysis of miRNA and mRNA paired expression profiling of primary fibroblast derived from diabetic foot ulcers reveals multiple impaired cellular functions.

MiR-21 and miR-205 are induced in invasive cutaneous squamous cell carcinomas.

Stress Signals, Mediated by Membranous Glucocorticoid Receptor, Activate PLC/PKC/GSK-3β/β-catenin Pathway to Inhibit Wound Closure.

Topical mevastatin promotes wound healing by inhibiting the transcription factor c-Myc via the glucocorticoid receptor and the long non-coding RNA Gas5.

Optical coherence tomography for assessment of epithelialization in a human ex vivo wound model.

Staphylococcus aureus Triggers Induction of miR-15B-5P to Diminish DNA Repair and Deregulate Inflammatory Response in Diabetic Foot Ulcers.

Probiotics or pro-healers: the role of beneficial bacteria in tissue repair.

Descriptive vs mechanistic scientific approach to study wound healing and its inhibition: Is there a value of translational research involving human subjects?

Novel mevalonate kinase missense mutation in a patient with disseminated superficial actinic porokeratosis.

Differentiation of diabetic foot ulcer-derived induced pluripotent stem cells reveals distinct cellular and tissue phenotypes.

Single cell analyses reveal specific distribution of anti-bacterial molecule Perforin-2 in human skin and its modulation by wounding and Staphylococcus aureus infection.

A tractable, simplified ex vivo human skin model of wound infection.

Preclinical evaluation of a novel silver gelling fiber dressing on Pseudomonas aeruginosa in a porcine wound infection model.

Pharmacological and Genetic Inhibition of Caveolin-1 Promotes Epithelialization and Wound Closure.

Mevastatin promotes healing by targeting caveolin-1 to restore EGFR signaling.

A bioengineered living cell construct activates metallothionein/zinc/MMP8 and inhibits TGFβ to stimulate remodeling of fibrotic venous leg ulcers.

Reply to "Questioning the use of an acute porcine wound model to assess anti-biofilm activity of dressings".

Skin Microbiota and its Interplay with Wound Healing.

Deregulated immune cell recruitment orchestrated by FOXM1 impairs human diabetic wound healing.

Boosts Innate Immune Response by Activation of Gamma Delta T Cells and Induction of Perforin-2 in Human Skin.

Intracellular escape strategies of Staphylococcus aureus in persistent cutaneous infections.

Correlates of injection-related wounds and skin infections amongst persons who inject drugs and use a syringe service programme: A single center study.

Epigenetic regulation of cellular functions in wound healing.

Wounds and Skin and Soft Tissue Infections in People Who Inject Drugs and the Utility of Syringe Service Programs in Their Management.

Cellular reprogramming of diabetic foot ulcer fibroblasts triggers pro-healing miRNA-mediated epigenetic signature.

Glucocorticoid-mediated induction of caveolin-1 disrupts cytoskeletal organization, inhibits cell migration and re-epithelialization of non-healing wounds.

Corrigendum: Boosts Innate Immune Response by Activation of Gamma Delta T Cells and Induction of Perforin-2 in Human Skin.

Intracellular Staphylococcus aureus triggers pyroptosis and contributes to inhibition of healing due to perforin-2 suppression.

Dichotomous role of miR193b-3p in diabetic foot ulcers maintains inhibition of healing and suppression of tumor formation.

FOXM1 network in association with TREM1 suppression regulates NET formation in diabetic foot ulcers.

Integrative small and long RNA omics analysis of human healing and nonhealing wounds discovers cooperating microRNAs as therapeutic targets.

Innate immunity and microbial dysbiosis in hidradenitis suppurativa - vicious cycle of chronic inflammation.

Urine-derived exosomes from individuals with IPF carry pro-fibrotic cargo.

Chronic wound microenvironment mediates selection of biofilm-forming multi drug resistant with capability to impair healing.

Novel Diagnostic Technologies and Therapeutic Approaches Targeting Chronic Wound Biofilms and Microbiota.

Cutaneous changes in diabetic patients: Primed for aberrant healing?

Calreticulin: a multifunctional protein with potential therapeutic applications for chronic wounds.

Spatial Distribution and Functional Impact of Human Scalp Hair Follicle Microbiota.

An Optimized and Advanced Algorithm for the Quantification of Immunohistochemical Biomarkers in Keratinocytes.

Postbiotic lactobacilli induce cutaneous antimicrobial response and restore the barrier to inhibit the intracellular invasion of Staphylococcus aureus in vitro and ex vivo.

Multi-drug resistant Staphylococcus epidermidis from chronic wounds impair healing in human wound model.

Tailored Bioengineering and Nanomedicine Strategies for Sex-Specific Healing of Chronic Wounds.

The Wound Reporting in Animal and Human Preclinical Studies (WRAHPS) Guidelines.