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Division of Neurotoxicology
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Embryo culture-based generation of enhanced green fluorescent protein-transgenic mice.
Biochemical and biophysical research communications Apr, 2003 | Pubmed ID: 12684032
Androgen receptor acetylation site mutations cause trafficking defects, misfolding, and aggregation similar to expanded glutamine tracts.
The Journal of biological chemistry Feb, 2004 | Pubmed ID: 14670946
Sperm membrane dynamics assessed by changes in lectin fluorescence before and after capacitation.
Journal of andrology Sep-Oct, 2004 | Pubmed ID: 15292105
The unfolded protein response modulates toxicity of the expanded glutamine androgen receptor.
The Journal of biological chemistry Jun, 2005 | Pubmed ID: 15799970
Pharmacologic and genetic inhibition of hsp90-dependent trafficking reduces aggregation and promotes degradation of the expanded glutamine androgen receptor without stress protein induction.
Human molecular genetics Jun, 2006 | Pubmed ID: 16644868
Reorganization of mouse sperm lipid rafts by capacitation.
Molecular reproduction and development Dec, 2006 | Pubmed ID: 16897730
Brain region-specific neurodegenerative profiles showing the relative importance of amphetamine dose, hyperthermia, seizures, and the blood-brain barrier.
Annals of the New York Academy of Sciences Oct, 2008 | Pubmed ID: 18991857
Small ubiquitin-like modifier (SUMO) modification of the androgen receptor attenuates polyglutamine-mediated aggregation.
The Journal of biological chemistry Aug, 2009 | Pubmed ID: 19497852
Amphetamine and environmentally induced hyperthermia differentially alter the expression of genes regulating vascular tone and angiogenesis in the meninges and associated vasculature.
Synapse (New York, N.Y.) Oct, 2009 | Pubmed ID: 19582783
The mRNA expression and histological integrity in rat forebrain motor and sensory regions are minimally affected by acrylamide exposure through drinking water.
Toxicology and applied pharmacology Nov, 2009 | Pubmed ID: 19664650
Endoplasmic reticulum stress responses differ in meninges and associated vasculature, striatum, and parietal cortex after a neurotoxic amphetamine exposure.
Synapse (New York, N.Y.) Aug, 2010 | Pubmed ID: 20340164
National Center for Toxicological Research
John F. Bowyer1,
Monzy Thomas1,
Tucker A. Patterson1,
Nysia I. George2,
Jeffrey A. Runnells3,
Mark S. Levi1
1Division of Neurotoxicology, National Center for Toxicological Research,
2Division of Personalized Nutrition and Medicine, National Center for Toxicological Research,
3Office of Planning, Finance, and Information Technology, National Center for Toxicological Research
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