Kathleen A. Kelly

Chemokine expression patterns differ within anatomically distinct regions of the genital tract during Chlamydia trachomatis infection.

A Chlamydia trachomatis-specific Th2 clone does not provide protection against a genital infection and displays reduced trafficking to the infected genital mucosa.

Chlamydia trachomatis enhances the expression of matrix metalloproteinases in an in vitro model of the human fallopian tube infection.

Cellular immunity and Chlamydia genital infection: induction, recruitment, and effector mechanisms.

The infecting dose of Chlamydia muridarum modulates the innate immune response and ascending infection.

COX-2 inhibition affects growth rate of Chlamydia muridarum within epithelial cells.

Stimulation of the cytosolic receptor for peptidoglycan, Nod1, by infection with Chlamydia trachomatis or Chlamydia muridarum.

Antigen-specific immunoglobulin E+ B cells are preferentially localized within germinal centres.

Epithelial membrane protein 2 modulates infectivity of Chlamydia muridarum (MoPn).

Blockade of epithelial membrane protein 2 (EMP2) abrogates infection of Chlamydia muridarum murine genital infection model.

Identification of dendritic cell subsets responding to genital infection by Chlamydia muridarum.

Targeting vault nanoparticles to specific cell surface receptors.

The combination of the gastrointestinal integrin (α4β7) and selectin ligand enhances T-Cell migration to the reproductive tract during infection with Chlamydia trachomatis.

Two different homing pathways involving integrin β7 and E-selectin significantly influence trafficking of CD4 cells to the genital tract following Chlamydia muridarum infection.

A vault nanoparticle vaccine induces protective mucosal immunity.

Plasmacytoid dendritic cells modulate nonprotective T-cell responses to genital infection by Chlamydia muridarum.

Objectified body consciousness in relation to recovery from an eating disorder.

Phenotype and function of regulatory T cells in the genital tract.

Vault Nanocapsules as Adjuvants Favor Cell-Mediated over Antibody-Mediated Immune Responses following Immunization of Mice.