Michael R. Hoane

The window of opportunity for administration of magnesium therapy following focal brain injury is 24 h but is task dependent in the rat.

No detectable analgesic effects in the formalin test even with one million bovine adrenal chromaffin cells.

The behavioral effects of magnesium therapy on recovery of function following bilateral anterior medial cortex lesions in the rat.

Treatment with vitamin B3 improves functional recovery and reduces GFAP expression following traumatic brain injury in rats.

Transplantation of neuronal and glial precursors dramatically improves sensorimotor function but not cognitive function in the traumatically injured brain.

Middle age increases tissue vulnerability and impairs sensorimotor and cognitive recovery following traumatic brain injury in the rat.

Magnesium therapy and recovery of function in experimental models of brain injury and neurodegenerative disease.

Treatment with magnesium improves reference memory but not working memory while reducing GFAP expression following traumatic brain injury.

Administration of riboflavin improves behavioral outcome and reduces edema formation and glial fibrillary acidic protein expression after traumatic brain injury.

Administration of raloxifene reduces sensorimotor and working memory deficits following traumatic brain injury.

Magnesium and riboflavin combination therapy following cortical contusion injury in the rat.

Nicotinamide reduces acute cortical neuronal death and edema in the traumatically injured brain.

Nicotinamide treatment reduces behavioral impairments and provides cortical protection after fluid percussion injury in the rat.

The effects of nicotinamide on apoptosis and blood-brain barrier breakdown following traumatic brain injury.

The novel apolipoprotein E-based peptide COG1410 improves sensorimotor performance and reduces injury magnitude following cortical contusion injury.

Nicotinamide treatment provides acute neuroprotection and GFAP regulation following fluid percussion injury.

Assessment of cognitive function following magnesium therapy in the traumatically injured brain.

Magnesium dietary manipulation and recovery of function following controlled cortical damage in the rat.

COG1410 improves cognitive performance and reduces cortical neuronal loss in the traumatically injured brain.

Strain differences in response to traumatic brain injury in Long-Evans compared to Sprague-Dawley rats.

The effects of hypertonic saline and nicotinamide on sensorimotor and cognitive function following cortical contusion injury in the rat.

Variation in chronic nicotinamide treatment after traumatic brain injury can alter components of functional recovery independent of histological damage.

Pyridoxine administration improves behavioral and anatomical outcome after unilateral contusion injury in the rat.

COG1410, an apolipoprotein E-based peptide, improves cognitive performance and reduces cortical loss following moderate fluid percussion injury in the rat.

Preclinical efficacy testing in middle-aged rats: nicotinamide, a novel neuroprotectant, demonstrates diminished preclinical efficacy after controlled cortical impact.

The effects of a high-fat sucrose diet on functional outcome following cortical contusion injury in the rat.

Continuous nicotinamide administration improves behavioral recovery and reduces lesion size following bilateral frontal controlled cortical impact injury.

The effect of progesterone dose on gene expression after traumatic brain injury.

Chronic folic acid administration confers no treatment effects in either a high or low dose following unilateral controlled cortical impact injury in the rat.

A discrimination task used as a novel method of testing decision-making behavior following traumatic brain injury.

A Comparison of the Effects of Nicotinamide and Progesterone on Functional Recovery of Cognitive Behavior following Cortical Contusion Injury in the Rat.