Laurence Ris

Neuronal deficiency of presenilin 1 inhibits amyloid plaque formation and corrects hippocampal long-term potentiation but not a cognitive defect of amyloid precursor protein [V717I] transgenic mice.

Effect of labyrinthectomy on the spike generator of vestibular neurons in the guinea pig.

Properties of neurons from the rat medial vestibular nucleus in microexplant culture.

Long-term plasticity of ipsilesional medial vestibular nucleus neurons after unilateral labyrinthectomy.

Capacitative calcium entry induces hippocampal long term potentiation in the absence of presenilin-1.

Labyrinthectomy changes T-type calcium channels in vestibular neurones of the guinea pig.

Loss of Ca2+/calmodulin kinase kinase beta affects the formation of some, but not all, types of hippocampus-dependent long-term memory.

Is there a role of the cyclin-dependent kinase 5 activator p25 in Alzheimer's disease?

The characteristics of LTP induced in hippocampal slices are dependent on slice-recovery conditions.

Ca2+/calmodulin kinase kinase alpha is dispensable for brain development but is required for distinct memories in male, though not in female, mice.

Tyrosine phosphorylation of rabphilin during long-lasting long-term potentiation.

Synapse specificity of long-term potentiation breaks down with aging.

Metaplastic effect of apamin on LTP and paired-pulse facilitation.

Involvement of hyperpolarization-activated cation channels in synaptic modulation.

Synaptic capture-mediated long-lasting long-term potentiation is strongly dependent on mRNA translation.

Late phase of L-LTP elicited in isolated CA1 dendrites cannot be transferred by synaptic capture.

Properties of contextual memory formed in the absence of αCaMKII autophosphorylation.

Mechanism for long-term memory formation when synaptic strengthening is impaired.

Long-lasting LTP requires neither repeated trains for its induction nor protein synthesis for its development.

Neurological characterization of mice deficient in GSK3α highlight pleiotropic physiological functions in cognition and pathological activity as Tau kinase.