Mingyu Liang

Renal medullary genes in salt-sensitive hypertension: a chromosomal substitution and cDNA microarray study.

Insights into Dahl salt-sensitive hypertension revealed by temporal patterns of renal medullary gene expression.

Gene expression reveals vulnerability to oxidative stress and interstitial fibrosis of renal outer medulla to nonhypertensive elevations of ANG II.

Quantitative assessment of the importance of dye switching and biological replication in cDNA microarray studies.

High throughput gene expression profiling: a molecular approach to integrative physiology.

Effect of high glucose on gene expression in mesangial cells: upregulation of the thiol pathway is an adaptational response.

Identification of persistently altered gene expression in the kidney after functional recovery from ischemic acute renal failure.

Tissue-specific transcriptome responses in rats with early streptozotocin-induced diabetes.

Transcriptome analysis and kidney research: toward systems biology.

Physiology and genomics: toward systems biology.

NADPH oxidase in the renal medulla causes oxidative stress and contributes to salt-sensitive hypertension in Dahl S rats.

Effect of sodium delivery on superoxide and nitric oxide in the medullary thick ascending limb.

The application of microarray analysis to pediatric diseases.

Thiol-related genes in diabetic complications: a novel protective role for endogenous thioredoxin 2.

Renal interstitial corticosterone and 11-dehydrocorticosterone in conscious rats.

Integrative pathway knowledge bases as a tool for systems molecular medicine.

Suppression of 11beta-hydroxysteroid dehydrogenase type 1 with RNA interference substantially attenuates 3T3-L1 adipogenesis.

MicroRNA-target pairs in the rat kidney identified by microRNA microarray, proteomic, and bioinformatic analysis.

Renal regional proteomes in young Dahl salt-sensitive rats.

High perfusion pressure accelerates renal injury in salt-sensitive hypertension.

Molecular networks in Dahl salt-sensitive hypertension based on transcriptome analysis of a panel of consomic rats.

Glucocorticoid response elements and 11 beta-hydroxysteroid dehydrogenases in the regulation of endothelial nitric oxide synthase expression.

Renal medullary 11 beta-hydroxysteroid dehydrogenase type 1 in Dahl salt-sensitive hypertension.

MicroRNA: a new frontier in kidney and blood pressure research.

MicroRNA: a new entrance to the broad paradigm of systems molecular medicine.

Novel role of fumarate metabolism in dahl-salt sensitive hypertension.

A novel amiloride-sensitive h+ transport pathway mediates enhanced superoxide production in thick ascending limb of salt-sensitive rats, not na+/h+ exchange.

Dynamic convergence and divergence of renal genomic and biological pathways in protection from Dahl salt-sensitive hypertension.

Renal medullary microRNAs in Dahl salt-sensitive rats: miR-29b regulates several collagens and related genes.

MicroRNA-target pairs in human renal epithelial cells treated with transforming growth factor beta 1: a novel role of miR-382.

Narrowing a region on rat chromosome 13 that protects against hypertension in Dahl SS-13BN congenic strains.

Hypertension as a mitochondrial and metabolic disease.

MiR-382 targeting of kallikrein 5 contributes to renal inner medullary interstitial fibrosis.

The miR-29 family: genomics, cell biology, and relevance to renal and cardiovascular injury.

Translational study of microRNAs and its application in kidney disease and hypertension research.

Increased expression of NAD(P)H oxidase subunit p67(phox) in the renal medulla contributes to excess oxidative stress and salt-sensitive hypertension.

Report of the National Heart, Lung, and Blood Institute Working Group on epigenetics and hypertension.

Delayed ischemic preconditioning contributes to renal protection by upregulation of miR-21.

Mitochondrial proteomic analysis reveals deficiencies in oxygen utilization in medullary thick ascending limb of Henle in the Dahl salt-sensitive rat.

Increased proliferative cells in the medullary thick ascending limb of the loop of Henle in the Dahl salt-sensitive rat.

MicroRNAs contribute to the maintenance of cell-type-specific physiological characteristics: miR-192 targets Na+/K+-ATPase β1.

miR-29c is downregulated in renal interstitial fibrosis in humans and rats and restored by HIF-α activation.

Characterization of human plasma-derived exosomal RNAs by deep sequencing.

miR-21 contributes to xenon-conferred amelioration of renal ischemia-reperfusion injury in mice.

Intermittent exposure to xenon protects against gentamicin-induced nephrotoxicity.

Epigenomics of hypertension.

Targeting the endothelial progenitor cell surface proteome to identify novel mechanisms that mediate angiogenic efficacy in a rodent model of vascular disease.

Characteristics of MicroRNAs Enriched in Specific Cell Types and Primary Tissue Types in Solid Organs.