Dennis Ladage

Cardio-Selective Beta-Blocker: Pharmacological Evidence and Their Influence on Exercise Capacity.

Temporal changes of strain parameters in the progress of chronic ischemia: with comparison to transmural infarction.

Crataegus special extract WS 1442 induces an endothelium-dependent, NO-mediated vasorelaxation via eNOS-phosphorylation at serine 1177.

Basic fibroblast growth factor controls migration in human mesenchymal stem cells.

Mesenchymal stem cells transmigrate over the endothelial barrier.

Mechanisms underlying nebivolol-induced endothelial nitric oxide synthase activation in human umbilical vein endothelial cells.

Mesenchymal stem cells induce endothelial activation via paracine mechanisms.

Characterisation of the interaction between circulating and in vitro cultivated endothelial progenitor cells and the endothelial barrier.

[Third generation beta-blockers: current state of research on vasodilating beta-blockers].

Hyperaldosteronism is associated with a decrease in number and altered growth factor expression of endothelial progenitor cells in rats.

SERCA2a gene transfer enhances eNOS expression and activity in endothelial cells.

Nebivolol lowers blood pressure and increases weight loss in patients with hypertension and diabetes in regard to age.

Multimodality imaging of chronic ischemia.

Method of gene delivery in large animal models of cardiovascular diseases.

Rescuing the failing heart by targeted gene transfer.

缺血性心肌病猪临床前期模型的发展。

Staged approach prevents spinal cord injury in hybrid surgical-endovascular thoracoabdominal aortic aneurysm repair: an experimental model.

Aldosterone induces electrical remodeling independent of hypertension.

Neutralizing antibodies against AAV serotypes 1, 2, 6, and 9 in sera of commonly used animal models.

Inhibition of PKCα/β with ruboxistaurin antagonizes heart failure in pigs after myocardial infarction injury.

Concomitant Intravenous Nitroglycerin With Intracoronary Delivery of AAV1.SERCA2a Enhances Gene Transfer in Porcine Hearts.

Assessing left ventricular systolic dysfunction after myocardial infarction: are the ejection fraction and the dP/dt maximum complementary or redundant?