Maria Kost-Alimova

Optimisation of the degenerate oligonucleotide primed PCR (DOP-PCR) for capillary thermocycler.

Mouse cytosolic and mitochondrial deoxyribonucleotidases: cDNA cloning of the mitochondrial enzyme, gene structures, chromosomal mapping and comparison with the human orthologs.

Comparative human/murine sequence analysis of the common eliminated region 1 from human 3p21.3.

Consistent downregulation of human lactoferrin gene, in the common eliminated region 1 on 3p21.3, following tumor growth in severe combined immunodeficient (SCID) mice.

Coincidence of synteny breakpoints with malignancy-related deletions on human chromosome 3.

Microcell-mediated chromosome transfer provides evidence that polysomy promotes structural instability in tumor cell chromosomes through asynchronous replication and breakage within late-replicating regions.

CGH analysis of familial non-BRCA1/BRCA2 breast tumors and mutation screening of a candidate locus on chromosome 17q11.2-12.

[Identification of 3q21q26 syndrome by "multipoint" interphase FISH analyses in childhood myeloid leukemia].

Jumping translocation of 17q11 approximately qter and 3q25 approximately q28 duplication in a variant Philadelphia t(9;14;22)(q34;q32;q11) in a childhood chronic myelogenous leukemia.

Multipoint interphase FISH analysis of chromosome 3 abnormalities in 28 childhood AML patients.

Down regulation of 3p genes, LTF, SLC38A3 and DRR1, upon growth of human chromosome 3-mouse fibrosarcoma hybrids in severe combined immunodeficiency mice.

CHCHD7-PLAG1 and TCEA1-PLAG1 gene fusions resulting from cryptic, intrachromosomal 8q rearrangements in pleomorphic salivary gland adenomas.

Modeling non-random deletions in cancer.

Multipoint interphase FISH in childhood T-acute lymphoblastic leukemia detects subpopulations that carry different chromosome 3 aberrations.

Array-CGH and multipoint FISH to decode complex chromosomal rearrangements.

Mandatory chromosomal segment balance in aneuploid tumor cells.

Segmental duplications and evolutionary plasticity at tumor chromosome break-prone regions.

Telomerase reactivation reverses tissue degeneration in aged telomerase-deficient mice.

Telomere dysfunction induces metabolic and mitochondrial compromise.

Lenalidomide targets clonogenic side population in multiple myeloma: pathophysiologic and clinical implications.

Antitelomerase therapy provokes ALT and mitochondrial adaptive mechanisms in cancer.

Telomerase reactivation following telomere dysfunction yields murine prostate tumors with bone metastases.

Role of telomere dysfunction in cardiac failure in Duchenne muscular dystrophy.

Oncogene ablation-resistant pancreatic cancer cells depend on mitochondrial function.

Telomere dysfunction suppresses multiple endocrine neoplasia in mice.