Sarah K. England

Molecular diversity of vascular potassium channel isoforms.

17beta-Estradiol upregulates distinct maxi-K channel transcripts in mouse uterus.

Myometrial maxi-K channel beta1 subunit modulation during pregnancy and after 17beta-estradiol stimulation.

Detection and implications of potassium channel alterations.

Kv channel subunits that contribute to voltage-gated K+ current in renal vascular smooth muscle.

Voltage-gated K+ channels in rat small cerebral arteries: molecular identity of the functional channels.

Estradiol binding to maxi-K channels induces their down-regulation via proteasomal degradation.

Abnormal coronary function in mice deficient in alpha1H T-type Ca2+ channels.

Maxi-K channels localize to caveolae in human myometrium: a role for an actin-channel-caveolin complex in the regulation of myometrial smooth muscle K+ current.

Translocation of an endoproteolytically cleaved maxi-K channel isoform: mechanisms to induce human myometrial cell repolarization.

The antibacterial activity of human neutrophils and eosinophils requires proton channels but not BK channels.

RhoA activation contributes to sex differences in vascular contractions.

Potassium channels and uterine function.

Overexpression of SK3 channels dampens uterine contractility to prevent preterm labor in mice.

Nardilysin convertase regulates the function of the maxi-K channel isoform mK44 in human myometrium.

Disruption of the maxi-K-caveolin-1 interaction alters current expression in human myometrial cells.

The contribution of Kv7 channels to pregnant mouse and human myometrial contractility.

In vivo measurement of intrauterine pressure by telemetry: a new approach for studying parturition in mouse models.

SK3 channel expression during pregnancy is regulated through estrogen and Sp factor-mediated transcriptional control of the KCNN3 gene.

Single-nucleotide polymorphisms in the KCNN3 gene associate with preterm birth.

Altered contribution of RhoA/Rho kinase signaling in contractile activity of myometrium in leptin receptor-deficient mice.

Effects of progesterone treatment on expression of genes involved in uterine quiescence.

Overexpression of the SK3 channel alters vascular remodeling during pregnancy, leading to fetal demise.

RhoA localization with caveolin-1 regulates vascular contractions to serotonin.

PPARγ regulates resistance vessel tone through a mechanism involving RGS5-mediated control of protein kinase C and BKCa channel activity.

N-terminal isoforms of the large-conductance Ca²⁺-activated K⁺ channel are differentially modulated by the auxiliary β1-subunit.

BK channels regulate myometrial contraction by modulating nuclear translocation of NF-κB.

The inwardly rectifying K+ channel KIR7.1 controls uterine excitability throughout pregnancy.

FSH receptor (FSHR) expression in human extragonadal reproductive tissues and the developing placenta, and the impact of its deletion on pregnancy in mice.

Functional insights into modulation of BKCa channel activity to alter myometrial contractility.

Reversibly switchable fluorescence microscopy with enhanced resolution and image contrast.

Factors associated with higher oxytocin requirements in labor.

The SK3 channel promotes placental vascularization by enhancing secretion of angiogenic factors.

BKCa channel inhibitor modulates the tumorigenic ability of hormone-independent breast cancer cells via the Wnt pathway.