William D. Phillips

GABARAP and GABA(A) receptor clustering.

Neuregulin potentiates agrin-induced acetylcholine receptor clustering in myotubes.

Increased ratio of rapsyn to ACh receptor stabilizes postsynaptic receptors at the mouse neuromuscular synapse.

Bose-Einstein condensates and precision measurements.

Neural agrin: a synaptic stabiliser.

Developmental increase in the amount of rapsyn per acetylcholine receptor promotes postsynaptic receptor packing and stability.

Controlled exchange interaction between pairs of neutral atoms in an optical lattice.

Anti-MuSK patient antibodies disrupt the mouse neuromuscular junction.

Neural agrin increases postsynaptic ACh receptor packing by elevating rapsyn protein at the mouse neuromuscular synapse.

TRPC1 binds to caveolin-3 and is regulated by Src kinase - role in Duchenne muscular dystrophy.

Multiphoton magnetooptical trap.

Muscle specific kinase: organiser of synaptic membrane domains.

Pulsed Sisyphus scheme for laser cooling of atomic (anti)hydrogen.

Ultracold atoms and precise time standards.

Caveolae respond to cell stretch and contribute to stretch-induced signaling.

Neuregulin-1 potentiates agrin-induced acetylcholine receptor clustering through muscle-specific kinase phosphorylation.

Observation of a superfluid Hall effect.

Muscle specific kinase autoantibodies cause synaptic failure through progressive wastage of postsynaptic acetylcholine receptors.

Partial-transfer absorption imaging: a versatile technique for optimal imaging of ultracold gases.

Migration of resident cardiac stem cells in myocardial infarction.

Pyridostigmine but not 3,4-diaminopyridine exacerbates ACh receptor loss and myasthenia induced in mice by muscle-specific kinase autoantibody.

Profile of David Wineland and Serge Haroche, 2012 Nobel laureates in physics.

Sequence of age-associated changes to the mouse neuromuscular junction and the protective effects of voluntary exercise.

Effects of the ß2-adrenoceptor agonist, albuterol, in a mouse model of anti-MuSK myasthenia gravis.

Hysteresis in a quantized superfluid 'atomtronic' circuit.

Muscle-specific kinase (MuSK) autoantibodies suppress the MuSK pathway and ACh receptor retention at the mouse neuromuscular junction.

Clinical and scientific aspects of muscle-specific tyrosine kinase-related myasthenia gravis.