Yingjin Wang

Albumin resuscitation increases cardiomyocyte contractility and decreases nitric oxide synthase II expression in rat endotoxemia.

Toll-like receptor stimulation in cardiomyoctes decreases contractility and initiates an NF-kappaB dependent inflammatory response.

Albumin resuscitation improves ventricular contractility and myocardial tissue oxygenation in rat endotoxemia.

S100A8 and S100A9 mediate endotoxin-induced cardiomyocyte dysfunction via the receptor for advanced glycation end products.

Vasopressin decreases sepsis-induced pulmonary inflammation through the V2R.

Low-dose vasopressin infusion results in increased mortality and cardiac dysfunction following ischemia-reperfusion injury in mice.

Extracellular heat shock protein 70 induces cardiomyocyte inflammation and contractile dysfunction via TLR2.

Notch-dependent regulation of the ischemic vasodilatory response--brief report.