Nathaniel J. Szewczyk

Activation of Ras and the mitogen-activated protein kinase pathway promotes protein degradation in muscle cells of Caenorhabditis elegans.

Activated EGL-15 FGF receptor promotes protein degradation in muscles of Caenorhabditis elegans.

Chemically defined medium and Caenorhabditis elegans.

Surviving atmospheric spacecraft breakup.

Signal-transduction networks and the regulation of muscle protein degradation.

Caenorhabditis elegans survives atmospheric breakup of STS-107, space shuttle Columbia.

Decreased expression of myogenic transcription factors and myosin heavy chains in Caenorhabditis elegans muscles developed during spaceflight.

Delayed development and lifespan extension as features of metabolic lifestyle alteration in C. elegans under dietary restriction.

Opposed growth factor signals control protein degradation in muscles of Caenorhabditis elegans.

unc-94 encodes a tropomodulin in Caenorhabditis elegans.

Genomic response of the nematode Caenorhabditis elegans to spaceflight.

Biochemical and Molecular Biological Analyses of space-flown nematodes in Japan, the First International Caenorhabditis elegans Experiment (ICE-First).

The glutaredoxin GLRX-21 functions to prevent selenium-induced oxidative stress in Caenorhabditis elegans.

The effectiveness of RNAi in Caenorhabditis elegans is maintained during spaceflight.

Identification and functional clustering of genes regulating muscle protein degradation from amongst the known C. elegans muscle mutants.

Remote automated multi-generational growth and observation of an animal in low Earth orbit.

Calpains mediate integrin attachment complex maintenance of adult muscle in Caenorhabditis elegans.

The next phase of life-sciences spaceflight research: Harnessing the power of functional genomics.

Selenium induces cholinergic motor neuron degeneration in Caenorhabditis elegans.

Genes down-regulated in spaceflight are involved in the control of longevity in Caenorhabditis elegans.

Using Multiple Phenotype Assays and Epistasis Testing to Enhance the Reliability of RNAi Screening and Identify Regulators of Muscle Protein Degradation.

Integrated control of protein degradation in C. elegans muscle.

Protective role of DNJ-27/ERdj5 in Caenorhabditis elegans models of human neurodegenerative diseases.

Focal adhesion kinase is required for IGF-I-mediated growth of skeletal muscle cells via a TSC2/mTOR/S6K1-associated pathway.

Evaluation of the Fluids Mixing Enclosure System for Life Science Experiments During a Commercial Caenorhabditis elegans Spaceflight Experiment.

Knockdown of the C. elegans kinome identifies kinases required for normal protein homeostasis, mitochondrial network structure, and sarcomere structure in muscle.

Spaceflight and ageing: reflecting on Caenorhabditis elegans in space.

A validation of the application of D(2)O stable isotope tracer techniques for monitoring day-to-day changes in muscle protein subfraction synthesis in humans.

The neurodegenerative effects of selenium are inhibited by FOXO and PINK1/PTEN regulation of insulin/insulin-like growth factor signaling in Caenorhabditis elegans.