Yuri Bozzi

Dopamine D2 receptor signaling controls neuronal cell death induced by muscarinic and glutamatergic drugs.

Effects of neurotrophins on synaptic protein expression in the visual cortex of dark-reared rats.

Intraocular delivery of BDNF following visual cortex lesion upregulates cytosolic branched chain aminotransferase (BCATc) in the rat dorsal lateral geniculate nucleus.

Simultaneous absence of dopamine D1 and D2 receptor-mediated signaling is lethal in mice.

Acute physiological response of mammalian central neurons to axotomy: ionic regulation and electrical activity.

BDNF down-regulates the caspase 3 pathway in injured geniculo-cortical neurones.

Antiepileptic effects of botulinum neurotoxin E.

Dopamine in neurotoxicity and neuroprotection: what do D2 receptors have to do with it?

Cytosolic branched chain aminotransferase (BCATc) mRNA is up-regulated in restricted brain areas of BDNF transgenic mice.

Expression of cytosolic branched chain aminotransferase (BCATc) mRNA in the developing mouse brain.

BoNT/E prevents seizure-induced activation of caspase 3 in the rat hippocampus.

BoNT/E prevents seizure-induced activation of caspase 3 in the rat hippocampus.

Increased levels of d-aspartate in the hippocampus enhance LTP but do not facilitate cognitive flexibility.

Activity-dependent phosphorylation of Ser187 is required for SNAP-25-negative modulation of neuronal voltage-gated calcium channels.

Botulinum neurotoxin E (BoNT/E) reduces CA1 neuron loss and granule cell dispersion, with no effects on chronic seizures, in a mouse model of temporal lobe epilepsy.

Fourth conference on epileptogenesis, May 23-26, 2007, Pisa, Italy.

Serotonin hyperinnervation abolishes seizure susceptibility in Otx2 conditional mutant mice.

Seizures increase importin-beta1 expression in NG2+ cells in the rat hippocampus.

Intrahippocampal infusion of botulinum neurotoxin E (BoNT/E) reduces spontaneous recurrent seizures in a mouse model of mesial temporal lobe epilepsy.

Impaired neurogenesis, learning and memory and low seizure threshold associated with loss of neural precursor cell survivin.

Kainic acid-induced seizures activate GSK-3β in the hippocampus of D2R-/- mice.

Environmental enrichment reduces spontaneous seizures in the Q54 transgenic mouse model of temporal lobe epilepsy.

Cell signaling underlying epileptic behavior.

The role of GABAergic system in neurodevelopmental disorders: a focus on autism and epilepsy.

Epilepsy as a neurodevelopmental disorder.

The atypical homeoprotein Pbx1a participates in the axonal pathfinding of mesencephalic dopaminergic neurons.

Mutant mouse models of autism spectrum disorders.

Epileptiform activity and cognitive deficits in SNAP-25(+/-) mice are normalized by antiepileptic drugs.

Kainic acid-induced seizures modulate Akt (SER473) phosphorylation in the hippocampus of dopamine D2 receptor knockout mice.

Loss of GABAergic neurons in the hippocampus and cerebral cortex of Engrailed-2 null mutant mice: implications for autism spectrum disorders.

The role of dopamine signaling in epileptogenesis.

Transcriptome profiling in engrailed-2 mutant mice reveals common molecular pathways associated with autism spectrum disorders.

Neural differentiation of pluripotent cells in 3D alginate-based cultures.

Noggin Expression in the Adult Retina Suggests a Conserved Role during Vertebrate Evolution.

Sorting of Sox1-GFP Mouse Embryonic Stem Cells Enhances Neuronal Identity Acquisition upon Factor-Free Monolayer Differentiation.

Altered GABAergic markers, increased binocularity and reduced plasticity in the visual cortex of Engrailed-2 knockout mice.

GH Dysfunction in Engrailed-2 Knockout Mice, a Model for Autism Spectrum Disorders.

Hippocampal dysregulation of neurofibromin-dependent pathways is associated with impaired spatial learning in engrailed 2 knock-out mice.

Synapses as therapeutic targets for autism spectrum disorders: an international symposium held in pavia on july 4th, 2014.

Early depolarizing GABA controls critical-period plasticity in the rat visual cortex.

New treatment perspectives in autism spectrum disorders.

Noggin-mediated Retinal Induction Reveals a Novel Interplay between BMP Inhibition, TGFβ and SHH Signaling.