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CD40 is expressed and functional on neuronal cells.

p35/Cdk5 pathway mediates soluble amyloid-beta peptide-induced tau phosphorylation in vitro.

CD40-CD40L interaction in Alzheimer's disease.

Role of CD40 ligand in amyloidosis in transgenic Alzheimer's mice.

Reduced Th1 and enhanced Th2 immunity after immunization with Alzheimer's beta-amyloid(1-42).

CD45 isoform alteration in CD4+ T cells as a potential diagnostic marker of Alzheimer's disease.

Increased vulnerability to focal ischemic brain injury in human apolipoprotein E4 knock-in mice.

Neuronal expression of CD22: novel mechanism for inhibiting microglial proinflammatory cytokine production.

CD45 isoform RB as a molecular target to oppose lipopolysaccharide-induced microglial activation in mice.

Augmented delayed infarct expansion and reactive astrocytosis after permanent focal ischemia in apolipoprotein E4 knock-in mice.

Behavioral effects of CD40-CD40L pathway disruption in aged PSAPP mice.

Toll-like receptor 3 mediates West Nile virus entry into the brain causing lethal encephalitis.

CD40 signaling regulates innate and adaptive activation of microglia in response to amyloid beta-peptide.

Modulation of astrocytic activation by arundic acid (ONO-2506) mitigates detrimental effects of the apolipoprotein E4 isoform after permanent focal ischemia in apolipoprotein E knock-in mice.

Interchromosomal associations between alternatively expressed loci.

Physiological regulation of the beta-amyloid precursor protein signaling domain by c-Jun N-terminal kinase JNK3 during neuronal differentiation.

Green tea epigallocatechin-3-gallate (EGCG) modulates amyloid precursor protein cleavage and reduces cerebral amyloidosis in Alzheimer transgenic mice.

T-cells in Alzheimer's disease.

The microglial "activation" continuum: from innate to adaptive responses.

Microglia recognize double-stranded RNA via TLR3.

ADAM10 activation is required for green tea (-)-epigallocatechin-3-gallate-induced alpha-secretase cleavage of amyloid precursor protein.

Arundic Acid ameliorates cerebral amyloidosis and gliosis in Alzheimer transgenic mice.

HSP70 enhances macrophage phagocytosis by interaction with lipid raft-associated TLR-7 and upregulating p38 MAPK and PI3K pathways.

Antiviral peptides targeting the west nile virus envelope protein.

Transcutaneous beta-amyloid immunization reduces cerebral beta-amyloid deposits without T cell infiltration and microhemorrhage.

NSAIDs for the chemoprevention of Alzheimer's disease.

Apoptotic hepatocyte DNA inhibits hepatic stellate cell chemotaxis via toll-like receptor 9.

Abrogation of macrophage migration inhibitory factor decreases West Nile virus lethality by limiting viral neuroinvasion.

Control of toll-like receptor 7 expression is essential to restrict autoimmunity and dendritic cell proliferation.

Toll-like receptor modulation of murine cerebral malaria is dependent on the genetic background of the host.

West Nile virus envelope protein inhibits dsRNA-induced innate immune responses.

Icam-1 participates in the entry of west nile virus into the central nervous system.

The stumpy gene is required for mammalian ciliogenesis.

Peripherally administered human umbilical cord blood cells reduce parenchymal and vascular beta-amyloid deposits in Alzheimer mice.

Defective p53 engagement after the induction of DNA damage in cells deficient in topoisomerase 3beta.

Flavonoid-mediated presenilin-1 phosphorylation reduces Alzheimer's disease beta-amyloid production.

Overexpression of human S100B exacerbates brain damage and periinfarct gliosis after permanent focal ischemia.

Blocking TGF-beta-Smad2/3 innate immune signaling mitigates Alzheimer-like pathology.

Drak2 contributes to West Nile virus entry into the brain and lethal encephalitis.

TGF-beta signaling in dendritic cells is a prerequisite for the control of autoimmune encephalomyelitis.

Primary cilia regulate hippocampal neurogenesis by mediating sonic hedgehog signaling.

Inflammaging as a prodrome to Alzheimer's disease.

Toll-like receptor 7 mitigates lethal West Nile encephalitis via interleukin 23-dependent immune cell infiltration and homing.

Approaches to determine expression of inflammatory cytokines.

Alternative Abeta immunotherapy approaches for Alzheimer's disease.

A role for the JAK-STAT1 pathway in blocking replication of HSV-1 in dendritic cells and macrophages.

A protective function for interleukin 17A in T cell-mediated intestinal inflammation.

CNS infiltration of peripheral immune cells: D-Day for neurodegenerative disease?

Overexpression of human S100B exacerbates cerebral amyloidosis and gliosis in the Tg2576 mouse model of Alzheimer's disease.

IL-10 signaling blockade controls murine West Nile virus infection.

Mutant presenilin-1 deregulated peripheral immunity exacerbates Alzheimer-like pathology.

Can peripheral leukocytes be used as Alzheimer's disease biomarkers?

Inflammation, immunity, and Alzheimer's disease.

Alzheimer's Disease Beyond Abeta.

Macrophages in Alzheimer's disease: the blood-borne identity.

Targeting S100B in Cerebral Ischemia and in Alzheimer's Disease.

Macrophage IL-12p70 signaling prevents HSV-1-induced CNS autoimmunity triggered by autoaggressive CD4+ Tregs.

CD45 deficiency drives amyloid-β peptide oligomers and neuronal loss in Alzheimer's disease mice.

The role of LAT in increased CD8+ T cell exhaustion in trigeminal ganglia of mice latently infected with herpes simplex virus 1.

Control of TH17 cells occurs in the small intestine.

Adaptive and innate transforming growth factor beta signaling impact herpes simplex virus 1 latency and reactivation.

Myeloid differentiation factor-2 interacts with Lyn kinase and is tyrosine phosphorylated following lipopolysaccharide-induced activation of the TLR4 signaling pathway.

Tannic acid is a natural β-secretase inhibitor that prevents cognitive impairment and mitigates Alzheimer-like pathology in transgenic mice.

Oxidized mitochondrial DNA activates the NLRP3 inflammasome during apoptosis.

Basic biology and mechanisms of neural ciliogenesis and the B9 family.

Rapid genetic targeting of pial surface neural progenitors and immature neurons by neonatal electroporation.

Microglia in Alzheimer's Disease: It's All About Context.

IL-22 signaling contributes to West Nile encephalitis pathogenesis.

Ferulic acid is a nutraceutical β-secretase modulator that improves behavioral impairment and alzheimer-like pathology in transgenic mice.

Increasing hematopoietic stem cell yield to develop mice with human immune systems.

Innate immunity in Alzheimer's disease: a complex affair.

A transgenic Alzheimer rat with plaques, tau pathology, behavioral impairment, oligomeric aβ, and frank neuronal loss.

Brain injury, neuroinflammation and Alzheimer's disease.

CD8α dendritic cells drive establishment of HSV-1 latency.

T-cell TGF-β signaling abrogation restricts medulloblastoma progression.

Methylene blue modulates β-secretase, reverses cerebral amyloidosis, and improves cognition in transgenic mice.

MyD88 is dispensable for cerebral amyloidosis and neuroinflammation in APP/PS1 transgenic mice.

The role of the immune system in neurodegenerative disorders: Adaptive or maladaptive?

Il10 deficiency rebalances innate immunity to mitigate Alzheimer-like pathology.

Spatial proximity of homologous alleles and long noncoding RNAs regulate a switch in allelic gene expression.

Neuroinflammation in Alzheimer's disease.

Characterization of cancer stem cells and primary cilia in medulloblastoma.

Innate Immunity Fights Alzheimer's Disease.

Increased Susceptibility of Humanized NSG Mice to Panton-Valentine Leukocidin and Staphylococcus aureus Skin Infection.

Multi-Shell Hybrid Diffusion Imaging (HYDI) at 7 Tesla in TgF344-AD Transgenic Alzheimer Rats.

CD8+ T Cells Play a Bystander Role in Mice Latently Infected with Herpes Simplex Virus 1.

Targeting innate immunity for neurodegenerative disorders of the central nervous system.

Interrelationship of Primary Virus Replication, Level of Latency, and Time to Reactivation in the Trigeminal Ganglia of Latently Infected Mice.

Amyloid Cascade into Clarity.

Interleukin-17A Promotes CD8+ T Cell Cytotoxicity To Facilitate West Nile Virus Clearance.

TLR8 Couples SOCS-1 and Restrains TLR7-Mediated Antiviral Immunity, Exacerbating West Nile Virus Infection in Mice.