Carol Imbriano

NF-Y recruitment of TFIID, multiple interactions with histone fold TAF(II)s.

Ternary complex formation between MADS-box transcription factors and the histone fold protein NF-YB.

Links between tumor suppressors: p53 is required for TGF-beta gene responses by cooperating with Smads.

Dynamic recruitment of NF-Y and histone acetyltransferases on cell-cycle promoters.

Transcriptional activation of the cyclin A gene by the architectural transcription factor HMGA2.

Cell cycle regulation of NF-YC nuclear localization.

Expression of the CCAAT-binding factor NF-Y in Caenorhabditis elegans.

Direct p53 transcriptional repression: in vivo analysis of CCAAT-containing G2/M promoters.

DNA damage promotes histone deacetylase 4 nuclear localization and repression of G2/M promoters, via p53 C-terminal lysines.

Dynamic recruitment of transcription factors and epigenetic changes on the ER stress response gene promoters.

A balance between NF-Y and p53 governs the pro- and anti-apoptotic transcriptional response.

Curcumin derivatives: molecular basis of their anti-cancer activity.

NF-YC complexity is generated by dual promoters and alternative splicing.

Specific inhibition of NF-Y subunits triggers different cell proliferation defects.

Structure-based design of potent aromatase inhibitors by high-throughput docking.

An acetylation-mono-ubiquitination switch on lysine 120 of H2B.

Newly synthesized curcumin derivatives: crosstalk between chemico-physical properties and biological activity.

The NF-Y/p53 liaison: well beyond repression.

Gain-of-function p53 mutants have widespread genomic locations partially overlapping with p63.

bis-Dehydroxy-Curcumin triggers mitochondrial-associated cell death in human colon cancer cells through ER-stress induced autophagy.

NF-Y coassociates with FOS at promoters, enhancers, repetitive elements, and inactive chromatin regions, and is stereo-positioned with growth-controlling transcription factors.

NF-Y activates genes of metabolic pathways altered in cancer cells.

Direct non transcriptional role of NF-Y in DNA replication.

Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition).

NF-YA splice variants have different roles on muscle differentiation.

NF-Y loss triggers p53 stabilization and apoptosis in HPV18-positive cells by affecting E6 transcription.

Transcriptionally regulated and nontoxic delivery of the hyperactive Sleeping Beauty Transposase.

Dynamic Phosphorylation of the Myocyte Enhancer Factor 2Cα1 Splice Variant Promotes Skeletal Muscle Regeneration and Hypertrophy.