Fengwu Li

Identification of novel Plasmodium gallinaceum zygote- and ookinete-expressed proteins as targets for blocking malaria transmission.

Monoclonal antibody against the Plasmodium falciparum chitinase, PfCHT1, recognizes a malaria transmission-blocking epitope in Plasmodium gallinaceum ookinetes unrelated to the chitinase PgCHT1.

Plasmodium ookinete-secreted proteins secreted through a common micronemal pathway are targets of blocking malaria transmission.

An anti-Chitinase malaria transmission-blocking single-chain antibody as an effector molecule for creating a Plasmodium falciparum-refractory mosquito.

Evidence-based annotation of the malaria parasite's genome using comparative expression profiling.

Apical surface expression of aspartic protease Plasmepsin 4, a potential transmission-blocking target of the plasmodium ookinete.

In vitro generation of Plasmodium falciparum ookinetes.

Engineered resistance to Plasmodium falciparum development in transgenic Anopheles stephensi.

Lack of molecular correlates of Plasmodium vivax ookinete development.

Algae-produced Pfs25 elicits antibodies that inhibit malaria transmission.

[An analysis of etiologies of fever of unknown origin in 372 patients].

Regulation of Neuroinflammation through Programed Death-1/Programed Death Ligand Signaling in Neurological Disorders.

Mutations in the P-type cation-transporter ATPase 4, PfATP4, mediate resistance to both aminopyrazole and spiroindolone antimalarials.

Alga-produced malaria transmission-blocking vaccine candidate Pfs25 formulated with a human use-compatible potent adjuvant induces high-affinity antibodies that block Plasmodium falciparum infection of mosquitoes.

SIRP/CD47 signaling in neurological disorders.

High-Throughput Assay and Discovery of Small Molecules that Interrupt Malaria Transmission.

Plasmodium falciparum ookinete expression of plasmepsin VII and plasmepsin X.

Open Source Drug Discovery with the Malaria Box Compound Collection for Neglected Diseases and Beyond.

A new clinically relevant model for intracranial atherosclerosis in rats.

Early rehabilitation aggravates brain damage after stroke via enhanced activation of nicotinamide adenine dinucleotide phosphate oxidase (NOX).

Omega-3 fatty acid supplement prevents development of intracranial atherosclerosis.

Enhanced apoptosis from early physical exercise rehabilitation following ischemic stroke.

Neuroprotection by Chlorpromazine and Promethazine in Severe Transient and Permanent Ischemic Stroke.

NOX Activation by Subunit Interaction and Underlying Mechanisms in Disease.

New Developments in the Pathophysiology, Workup, and Diagnosis of Dural Venous Sinus Thrombosis (DVST) and a Systematic Review of Endovascular Treatments.

Exercise rehabilitation immediately following ischemic stroke exacerbates inflammatory injury.

Erratum to: Neuroprotection by Chlorpromazine and Promethazine in Severe Transient and Permanent Ischemic Stroke.