Julio E. Ayala

Accessory elements, flanking DNA sequence, and promoter context play key roles in determining the efficacy of insulin and phorbol ester signaling through the malic enzyme and collagenase-1 AP-1 motifs.

Interaction of physiological mechanisms in control of muscle glucose uptake.

Considerations in the design of hyperinsulinemic-euglycemic clamps in the conscious mouse.

Chronic treatment with sildenafil improves energy balance and insulin action in high fat-fed conscious mice.

Glucose kinetics and exercise tolerance in mice lacking the GLUT4 glucose transporter.

Insulin action in the double incretin receptor knockout mouse.

Glucose metabolism in vivo in four commonly used inbred mouse strains.

The glucagon-like peptide-1 receptor regulates endogenous glucose production and muscle glucose uptake independent of its incretin action.

Skeletal muscle AMP-activated protein kinase is essential for the metabolic response to exercise in vivo.

NIH experiment in centralized mouse phenotyping: the Vanderbilt experience and recommendations for evaluating glucose homeostasis in the mouse.

Lost in translation.

Endothelial nitric oxide synthase is central to skeletal muscle metabolic regulation and enzymatic signaling during exercise in vivo.

Glucagon-like peptide-1 receptor knockout mice are protected from high-fat diet-induced insulin resistance.

Standard operating procedures for describing and performing metabolic tests of glucose homeostasis in mice.

The physiological regulation of glucose flux into muscle in vivo.

Diet-induced muscle insulin resistance is associated with extracellular matrix remodeling and interaction with integrin alpha2beta1 in mice.

Acute activation of central GLP-1 receptors enhances hepatic insulin action and insulin secretion in high-fat-fed, insulin resistant mice.

The nuclear receptor PPARβ/δ programs muscle glucose metabolism in cooperation with AMPK and MEF2.