Michael P. Feneley

The preload recruitable stroke work relationship as a measure of left ventricular contractile dysfunction in porcine cardiac allografts.

Lazaroid (U74389G)-supplemented cardioplegia: results of a double-blind, randomized, controlled trial in a porcine model of orthotopic heart transplantation.

The initial rate of troponin I release post-reperfusion reflects the effectiveness of myocardial protection during cardiac allograft preservation.

Functional evidence of reversible ischemic injury immediately after the sympathetic storm associated with experimental brain death.

Defects in nuclear structure and function promote dilated cardiomyopathy in lamin A/C-deficient mice.

Involvement of chymase-mediated angiotensin II generation in blood pressure regulation.

Mutations in cardiac T-box factor gene TBX20 are associated with diverse cardiac pathologies, including defects of septation and valvulogenesis and cardiomyopathy.

Safety and efficacy of consecutive cycles of granulocyte-colony stimulating factor, and an intracoronary CD133+ cell infusion in patients with chronic refractory ischemic heart disease: the G-CSF in angina patients with IHD to stimulate neovascularization (GAIN I) trial.

Effects of mechanical stress and carvedilol in lamin A/C-deficient dilated cardiomyopathy.

Neuregulin 1 sustains the gene regulatory network in both trabecular and nontrabecular myocardium.

Reproducibility of adenosine stress cardiovascular magnetic resonance in multi-vessel symptomatic coronary artery disease.

Nesprin-1 and actin contribute to nuclear and cytoskeletal defects in lamin A/C-deficient cardiomyopathy.

Evaluation of left ventricular enlargement as a marker of early disease in familial dilated cardiomyopathy.

Investigation of association between PFO complicated by cryptogenic stroke and a common variant of the cardiac transcription factor GATA4.

A randomised, placebo-controlled trial of carvedilol in early familial dilated cardiomyopathy.

Granulocyte colony stimulating factor in chronic angina to stimulate neovascularisation: a placebo controlled crossover trial.

RhoA/ROCK signaling and pleiotropic α1A-adrenergic receptor regulation of cardiac contractility.

Irreversible triggers for hypertrophic cardiomyopathy are established in the early postnatal period.

Snowflakes in the heart: an ultrasonic marker of severe hypercoagulability.

Pressure Overload by Transverse Aortic Constriction Induces Maladaptive Hypertrophy in a Titin-Truncated Mouse Model.

Transcatheter deployment of two atrial septal defect closure devices using 3-dimensional transoesophageal echocardiography guidance.

Pathophysiological Trends During Withdrawal of Life Support: Implications for Organ Donation After Circulatory Death.

Standardized echocardiographic assessment of cardiac function in normal adult zebrafish and heart disease models.

High-Frequency Echocardiography　- Transformative Clinical and Research Applications in Humans, Mice, and Zebrafish.

Orphan receptor GPR37L1 contributes to the sexual dimorphism of central cardiovascular control.

Cardiac hypertrophy limits infarct expansion after myocardial infarction in mice.

Endocardial TRPC-6 Channels Act as Atrial Mechanosensors and Load-Dependent Modulators of Endocardial/Myocardial Cross-Talk.

A-Band Titin Truncation in Zebrafish Causes Dilated Cardiomyopathy and Hemodynamic Stress Intolerance.

Gene-environment interaction impacts on heart development and embryo survival.

Platelet-derived growth factor-AB improves scar mechanics and vascularity after myocardial infarction.

Non-Invasive Quantification of Ventricular Contractility, Arterial Elastic Function and Ventriculo-Arterial Coupling from a Single Diagnostic Encounter Using Simultaneous Arterial Tonometry and Magnetic Resonance Imaging.

In vitro cell stretching technology (IsoStretcher) as an approach to unravel Piezo1-mediated cardiac mechanotransduction.

Pressure overload by suprarenal aortic constriction in mice leads to left ventricular hypertrophy without c-Kit expression in cardiomyocytes.

Cardiac Gq Receptors and Calcineurin Activation Are Not Required for the Hypertrophic Response to Mechanical Left Ventricular Pressure Overload.

The Ca-activated cation channel TRPM4 is a positive regulator of pressure overload-induced cardiac hypertrophy.

Modified N-linked glycosylation status predicts trafficking defective human Piezo1 channel mutations.

Involvement of GPR37L1 in murine blood pressure regulation and human cardiac disease pathophysiology.