Jerry E. Chipuk
Department of Oncological Sciences,
Department of Dermatology,
The Tisch Cancer Institute,
The Graduate School of Biological Sciences,
Department of Oncological Sciences, Department of Dermatology, The Tisch Cancer Institute, The Graduate School of Biological Sciences
Mount Sinai School of Medicine
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The androgen receptor represses transforming growth factor-beta signaling through interaction with Smad3.
The Journal of biological chemistry Jan, 2002 | Pubmed ID: 11707452
Identification and characterization of a novel rat ov-serpin family member, trespin.
The Journal of biological chemistry Jul, 2002 | Pubmed ID: 11986314
p53's believe it or not: lessons on transcription-independent death.
Journal of clinical immunology Sep, 2003 | Pubmed ID: 14601643
Pharmacologic activation of p53 elicits Bax-dependent apoptosis in the absence of transcription.
Cancer cell Nov, 2003 | Pubmed ID: 14667504
Direct activation of Bax by p53 mediates mitochondrial membrane permeabilization and apoptosis.
Science (New York, N.Y.) Feb, 2004 | Pubmed ID: 14963330
Cytoplasmic p53: bax and forward.
Cell cycle (Georgetown, Tex.) Apr, 2004 | Pubmed ID: 15020844
Do inducers of apoptosis trigger caspase-independent cell death?
Nature reviews. Molecular cell biology Mar, 2005 | Pubmed ID: 15714200
BH3 domains of BH3-only proteins differentially regulate Bax-mediated mitochondrial membrane permeabilization both directly and indirectly.
Molecular cell Feb, 2005 | Pubmed ID: 15721256
PUMA couples the nuclear and cytoplasmic proapoptotic function of p53.
Science (New York, N.Y.) Sep, 2005 | Pubmed ID: 16151013
p53 and metabolism: Inside the TIGAR.
Cell Jul, 2006 | Pubmed ID: 16839873
Chemical inhibition of the mitochondrial division dynamin reveals its role in Bax/Bak-dependent mitochondrial outer membrane permeabilization.
Developmental cell Feb, 2008 | Pubmed ID: 18267088
How do BCL-2 proteins induce mitochondrial outer membrane permeabilization?
Trends in cell biology Apr, 2008 | Pubmed ID: 18314333
Apoptosis: Stabbed in the BAX.
Nature Oct, 2008 | Pubmed ID: 18948940
Mechanism of apoptosis induction by inhibition of the anti-apoptotic BCL-2 proteins.
Proceedings of the National Academy of Sciences of the United States of America Dec, 2008 | Pubmed ID: 19074266
Mitochondrial outer membrane proteins assist Bid in Bax-mediated lipidic pore formation.
Molecular biology of the cell Apr, 2009 | Pubmed ID: 19244344
PUMA cooperates with direct activator proteins to promote mitochondrial outer membrane permeabilization and apoptosis.
Cell cycle (Georgetown, Tex.) Sep, 2009 | Pubmed ID: 19652530
The BCL-2 family reunion.
Molecular cell Feb, 2010 | Pubmed ID: 20159550
PB1-F2 proteins from H5N1 and 20 century pandemic influenza viruses cause immunopathology.
PLoS pathogens , 2010 | Pubmed ID: 20661425
BH3 domains other than Bim and Bid can directly activate Bax/Bak.
The Journal of biological chemistry Jan, 2011 | Pubmed ID: 21041309
The Role of BH3-Only Proteins in Tumor Cell Development, Signaling, and Treatment.
Genes & cancer May, 2011 | Pubmed ID: 21901166
Born to be alive: a role for the BCL-2 family in melanoma tumor cell survival, apoptosis, and treatment.
Frontiers in oncology Oct, 2011 | Pubmed ID: 22268005
Sphingolipid metabolism cooperates with BAK and BAX to promote the mitochondrial pathway of apoptosis.
Cell Mar, 2012 | Pubmed ID: 22385963
GM-CSF controls nonlymphoid tissue dendritic cell homeostasis but is dispensable for the differentiation of inflammatory dendritic cells.
Immunity Jun, 2012 | Pubmed ID: 22749353
