Keith Nehrke

Molecular and functional characterization of a murine calcium-activated chloride channel expressed in smooth muscle.

Loss of hyperpolarization-activated Cl(-) current in salivary acinar cells from Clcn2 knockout mice.

The NHX family of Na+-H+ exchangers in Caenorhabditis elegans.

Molecular identification of Ca2+-activated K+ channels in parotid acinar cells.

Secretion and cell volume regulation by salivary acinar cells from mice lacking expression of the Clcn3 Cl- channel gene.

Altered GABAergic function accompanies hippocampal degeneration in mice lacking ClC-3 voltage-gated chloride channels.

Active K+ secretion through multiple KCa-type channels and regulation by IKCa channels in rat proximal colon.

The hSK4 (KCNN4) isoform is the Ca2+-activated K+ channel (Gardos channel) in human red blood cells.

Genome-wide search and identification of a novel gel-forming mucin MUC19/Muc19 in glandular tissues.

A reduction in intestinal cell pHi due to loss of the Caenorhabditis elegans Na+/H+ exchanger NHX-2 increases life span.

Alternative splicing of N- and C-termini of a C. elegans ClC channel alters gating and sensitivity to external Cl- and H+.

Physiological roles of the intermediate conductance, Ca2+-activated potassium channel Kcnn4.

GCK-3, a newly identified Ste20 kinase, binds to and regulates the activity of a cell cycle-dependent ClC anion channel.

The abts and sulp families of anion transporters from Caenorhabditis elegans.

Quantitative analysis of the voltage-dependent gating of mouse parotid ClC-2 chloride channel.

Altered gating and regulation of a carboxy-terminal ClC channel mutant expressed in the Caenorhabditis elegans oocyte.

Ste20-type kinases: evolutionarily conserved regulators of ion transport and cell volume.

Carboxy terminus splice variation alters ClC channel gating and extracellular cysteine reactivity.

Genetic hypercalciuric stone-forming rats.

Function of a STIM1 homologue in C. elegans: evidence that store-operated Ca2+ entry is not essential for oscillatory Ca2+ signaling and ER Ca2+ homeostasis.

Intracellular pH measurements in vivo using green fluorescent protein variants.

Oscillatory transepithelial H(+) flux regulates a rhythmic behavior in C. elegans.

The C. elegans mitochondrial K+(ATP) channel: a potential target for preconditioning.

Metabolic acidosis increases intracellular calcium in bone cells through activation of the proton receptor OGR1.

Loss of the apical V-ATPase a-subunit VHA-6 prevents acidification of the intestinal lumen during a rhythmic behavior in C. elegans.

A novel mitochondrial K(ATP) channel assay.

Mitochondrial fragmentation leads to intracellular acidification in Caenorhabditis elegans and mammalian cells.

The mitochondrial complex II and ATP-sensitive potassium channel interaction: quantitation of the channel in heart mitochondria.

A calcineurin homologous protein is required for sodium-proton exchange events in the C. elegans intestine.

Ischemic preconditioning: the role of mitochondria and aging.

SLO-2 is cytoprotective and contributes to mitochondrial potassium transport.

Regulation of acid-base transporters by reactive oxygen species following mitochondrial fragmentation.

Identification of a nuclear carbonic anhydrase in Caenorhabditis elegans.

Mitochondrial ATP-sensitive potassium channel activity and hypoxic preconditioning are independent of an inwardly rectifying potassium channel subunit in Caenorhabditis elegans.

Sex modifies genetic effects on residual variance in urinary calcium excretion in rat (Rattus norvegicus).

Effect of Caenorhabditis elegans age and genotype on horizontal gene transfer in intestinal bacteria.

miR-786 regulation of a fatty-acid elongase contributes to rhythmic calcium-wave initiation in C. elegans.

Bicarbonate modulates oxidative and functional damage in ischemia-reperfusion.

Physiological consequences of complex II inhibition for aging, disease, and the mKATP channel.

Analysis of Ca2+ signaling motifs that regulate proton signaling through the Na+/H+ exchanger NHX-7 during a rhythmic behavior in Caenorhabditis elegans.

Kir6.2 is not the mitochondrial KATP channel but is required for cardioprotection by ischemic preconditioning.

A non-cardiomyocyte autonomous mechanism of cardioprotection involving the SLO1 BK channel.

Anthranilate fluorescence marks a calcium-propagated necrotic wave that promotes organismal death in C. elegans.

Expression of the CHOP-inducible carbonic anhydrase CAVI-b is required for BDNF-mediated protection from hypoxia.