Pierre Hardy

PGE(2)-mediated eNOS induction in prolonged hypercapnia.

Pharmacokinetics and safety of the ketolide telithromycin in patients with renal impairment.

Redox-dependent effects of nitric oxide on microvascular integrity in oxygen-induced retinopathy.

New insights into the retinal circulation: inflammatory lipid mediators in ischemic retinopathy.

Dominant role for calpain in thromboxane-induced neuromicrovascular endothelial cytotoxicity.

Trans-arachidonic acids generated during nitrative stress induce a thrombospondin-1-dependent microvascular degeneration.

Inflammatory lipid mediators in ischemic retinopathy.

Hypercapnia prevents neovascularization via nitrative stress.

Activation of CD36 inhibits and induces regression of inflammatory corneal neovascularization.

Hyperoxic exposure leads to nitrative stress and ensuing microvascular degeneration and diminished brain mass and function in the immature subject.

Lysophosphatidic acid induces endothelial cell death by modulating the redox environment.

Lymphocytic microparticles inhibit angiogenesis by stimulating oxidative stress and negatively regulating VEGF-induced pathways.

trans-Arachidonic acids induce a heme oxygenase-dependent vasorelaxation of cerebral microvasculature.

Genetic ablation of CD36 induces age-related corneal neovascularization.

The succinate receptor GPR91 in neurons has a major role in retinal angiogenesis.

Interleukin-1 and ischemic brain injury in the newborn: development of a small molecule inhibitor of IL-1 receptor.

The role of lysophosphatidic acid receptor (LPA1) in the oxygen-induced retinal ganglion cell degeneration.

Hypoxia up-regulates CD36 expression and function via hypoxia-inducible factor-1- and phosphatidylinositol 3-kinase-dependent mechanisms.

Understanding ischemic retinopathies: emerging concepts from oxygen-induced retinopathy.

Sustained hypercapnia induces cerebral microvascular degeneration in the immature brain through induction of nitrative stress.

Low density lipoprotein receptor mediates anti-VEGF effect of lymphocyte T-derived microparticles in Lewis lung carcinoma cells.

Retinopathy of prematurity: understanding ischemic retinal vasculopathies at an extreme of life.

CD36 plays an important role in the clearance of oxLDL and associated age-dependent sub-retinal deposits.

Cortactin activation by FVIIa/tissue factor and PAR2 promotes endothelial cell migration.

Choroidal involution is a key component of oxygen-induced retinopathy.

Ghrelin modulates physiologic and pathologic retinal angiogenesis through GHSR-1a.

Role of receptor-mediated endocytosis in the antiangiogenic effects of human T lymphoblastic cell-derived microparticles.

Lymphocyte-derived microparticles induce bronchial epithelial cells' pro-inflammatory cytokine production and apoptosis.

Fatty acid receptor Gpr40 mediates neuromicrovascular degeneration induced by transarachidonic acids in rodents.

Microglia and interleukin-1β in ischemic retinopathy elicit microvascular degeneration through neuronal semaphorin-3A.

p75 neurotrophin receptor participates in the choroidal antiangiogenic and apoptotic effects of T-lymphocyte-derived microparticles.

Lymphocyte-derived microparticles induce apoptosis of airway epithelial cells through activation of p38 MAPK and production of arachidonic acid.

Anti-proliferative and anti-tumour effects of lymphocyte-derived microparticles are neither species- nor tumour-type specific.

Subcellular localization of coagulation factor II receptor-like 1 in neurons governs angiogenesis.

Retinal Neurons Curb Inflammation and Enhance Revascularization in Ischemic Retinopathies via Proteinase-Activated Receptor-2.