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Department of Biological Chemistry
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Na+ promotes the dissociation between Galpha GDP and Gbeta gamma, activating G protein-gated K+ channels.
The Journal of biological chemistry Feb, 2003 | Pubmed ID: 12488455
Mapping the Gbetagamma-binding sites in GIRK1 and GIRK2 subunits of the G protein-activated K+ channel.
The Journal of biological chemistry Aug, 2003 | Pubmed ID: 12743112
Galphai1 and Galphai3 differentially interact with, and regulate, the G protein-activated K+ channel.
The Journal of biological chemistry Apr, 2004 | Pubmed ID: 14963032
Gbetagamma-dependent and Gbetagamma-independent basal activity of G protein-activated K+ channels.
The Journal of biological chemistry Apr, 2005 | Pubmed ID: 15728579
Kinetic modeling of Na(+)-induced, Gbetagamma-dependent activation of G protein-gated K(+) channels.
Journal of molecular neuroscience : MN , 2005 | Pubmed ID: 15781962
Amplitude histogram-based method of analysis of patch clamp recordings that involve extreme changes in channel activity levels.
Journal of molecular neuroscience : MN Mar, 2009 | Pubmed ID: 18622586
Retrograde signaling in axonal regeneration.
Experimental neurology May, 2010 | Pubmed ID: 19699198
Axoplasm isolation from peripheral nerve.
Developmental neurobiology Feb, 2010 | Pubmed ID: 19885832
Signaling to transcription networks in the neuronal retrograde injury response.
Science signaling , 2010 | Pubmed ID: 20628157
Behavioral and other phenotypes in a cytoplasmic Dynein light intermediate chain 1 mutant mouse.
The Journal of neuroscience : the official journal of the Society for Neuroscience Apr, 2011 | Pubmed ID: 21471385
Axonal transcription factors signal retrogradely in lesioned peripheral nerve.
The EMBO journal Jan, 2012 | Pubmed ID: 22246183
Weizmann Institute of Science
Ida Rishal1,
Meir Rozenbaum1,
Mike Fainzilber1
1Department of Biological Chemistry, Weizmann Institute of Science
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