Pei-Lu Yi

The central serotonergic system mediates the analgesic effect of electroacupuncture on ZUSANLI (ST36) acupoints.

Kindling stimuli delivered at different times in the sleep-wake cycle.

Effects of electroacupuncture at 'Anmian (Extra)' acupoints on sleep activities in rats: the implication of the caud al nucleus tractus solitarius.

Gamma-aminobutyric acid (GABA) receptor mediates suanzaorentang, a traditional Chinese herb remedy, -induced sleep alteration.

Interleukin-1beta mediates sleep alteration in rats with rotenone-induced parkinsonism.

The involvement of serotonin receptors in suanzaorentang-induced sleep alteration.

Endogenous opiates in the nucleus tractus solitarius mediate electroacupuncture-induced sleep activities in rats.

TNF-NF-kappaB signaling mediates excessive somnolence in hemiparkinsonian rats.

Biphasic effects of baicalin, an active constituent of Scutellaria baicalensis Georgi, in the spontaneous sleep-wake regulation.

Effect of cannabidiol on sleep disruption induced by the repeated combination tests consisting of open field and elevated plus-maze in rats.

Kappa-opioid receptors in the caudal nucleus tractus solitarius mediate 100 hz electroacupuncture-induced sleep activities in rats.

Activation of GABAergic pathway by hypocretin in the median raphe nucleus (MRN) mediates stress-induced theta rhythm in rats.

Occurrence of epilepsy at different zeitgeber times alters sleep homeostasis differently in rats.

Electrical stimulation of left anterior thalamic nucleus with high-frequency and low-intensity currents reduces the rate of pilocarpine-induced epilepsy in rats.

Disruption of footshock-induced theta rhythms by stimulating median raphe nucleus reduces anxiety in rats.

Activation of amygdala opioid receptors by electroacupuncture of Feng-Chi (GB20) acupoints exacerbates focal epilepsy.

Amygdala opioid receptors mediate the electroacupuncture-induced deterioration of sleep disruptions in epilepsy rats.

Low-frequency electroacupuncture suppresses focal epilepsy and improves epilepsy-induced sleep disruptions.

Interleukin-1 receptor (IL-1R) mediates epilepsy-induced sleep disruption.