Paul Brehm

The Zebrafish motility mutant twitch once reveals new roles for rapsyn in synaptic function.

Optical measurements of presynaptic release in mutant zebrafish lacking postsynaptic receptors.

Increased neuromuscular activity causes axonal defects and muscular degeneration.

Persistent electrical coupling and locomotory dysfunction in the zebrafish mutant shocked.

Acetylcholine receptors direct rapsyn clusters to the neuromuscular synapse in zebrafish.

Tethering naturally occurring peptide toxins for cell-autonomous modulation of ion channels and receptors in vivo.

A mutation in serca underlies motility dysfunction in accordion zebrafish.

Paired motor neuron-muscle recordings in zebrafish test the receptor blockade model for shaping synaptic current.

An electrically coupled network of skeletal muscle in zebrafish distributes synaptic current.

Function of neuromuscular synapses in the zebrafish choline-acetyltransferase mutant bajan.

Synaptic homeostasis in a zebrafish glial glycine transporter mutant.

Distinct roles for two synaptotagmin isoforms in synchronous and asynchronous transmitter release at zebrafish neuromuscular junction.

An acetylcholine receptor lacking both γ and ε subunits mediates transmission in zebrafish slow muscle synapses.