Linda Brzustowicz

Linkage of familial schizophrenia to chromosome 13q32.

Location of a major susceptibility locus for familial schizophrenia on chromosome 1q21-q22.

Genetic insights into schizophrenia.

A major susceptibility locus for specific language impairment is located on 13q21.

Fine mapping of the schizophrenia susceptibility locus on chromosome 1q22.

Genome scan meta-analysis of schizophrenia and bipolar disorder, part II: Schizophrenia.

Linkage disequilibrium mapping of schizophrenia susceptibility to the CAPON region of chromosome 1q22.

Examination of potential overlap in autism and language loci on chromosomes 2, 7, and 13 in two independent samples ascertained for specific language impairment.

Increased expression in dorsolateral prefrontal cortex of CAPON in schizophrenia and bipolar disorder.

Support for the homeobox transcription factor gene ENGRAILED 2 as an autism spectrum disorder susceptibility locus.

Evaluation of a susceptibility gene for schizophrenia: genotype based meta-analysis of RGS4 polymorphisms from thirteen independent samples.

Association of synapsin 2 with schizophrenia in families of Northern European ancestry.

NOS1AP in schizophrenia.

Phenotype matters: the case for careful characterization of relevant traits.

Improvements to bead-based oligonucleotide ligation SNP genotyping assays.

Evidence for a role of the NOS1AP (CAPON) gene in schizophrenia and its clinical dimensions: an association study in a South American population isolate.

Identification of a schizophrenia-associated functional noncoding variant in NOS1AP.

NOS1AP regulates dendrite patterning of hippocampal neurons through a carboxypeptidase E-mediated pathway.

Autism-associated haplotype affects the regulation of the homeobox gene, ENGRAILED 2.

Copy number variations in schizophrenia: critical review and new perspectives on concepts of genetics and disease.

Childhood trauma and genetic factors in familial schizophrenia associated with the NOS1AP gene.

NOS1AP protein levels are altered in BA46 and cerebellum of patients with schizophrenia.

Reduced gray matter in the anterior cingulate gyrus in familial schizophrenia: a preliminary report.

Increasing genotype-phenotype model determinism: application to bivariate reading/language traits and epistatic interactions in language-impaired families.

Combined linkage and linkage disequilibrium analysis of a motor speech phenotype within families ascertained for autism risk loci.

Altered microRNA expression profiles in postmortem brain samples from individuals with schizophrenia and bipolar disorder.

Genetic covariation underlying reading, language and related measures in a sample selected for specific language impairment.

Validation of a cost-efficient multi-purpose SNP panel for disease based research.

Early environmental exposures influence schizophrenia expression even in the presence of strong genetic predisposition.

Gene × gene interaction in shared etiology of autism and specific language impairment.

miRNA-mediated risk for schizophrenia in 22q11.2 deletion syndrome.

Chronological changes in microRNA expression in the developing human brain.

A genome scan for loci shared by autism spectrum disorder and language impairment.

Revisiting schizophrenia linkage data in the NIMH Repository: reanalysis of regularized data across multiple studies.

Meta-analysis of repository data: impact of data regularization on NIMH schizophrenia linkage results.

Autism associated gene, engrailed2, and flanking gene levels are altered in post-mortem cerebellum.

The value of regenotyping older linkage data sets with denser marker panels.

MicroRNA Dysregulation, Gene Networks, and Risk for Schizophrenia in 22q11.2 Deletion Syndrome.

Nitric oxide synthase 1 adaptor protein, a protein implicated in schizophrenia, controls radial migration of cortical neurons.

Childhood maltreatment predicts allostatic load in adulthood.

Reply to: Reproducibility and Visual Inspection of Data.