Edward Glasscock

Genomic biomarkers of SUDEP in brain and heart.

Spontaneous seizures in Kcna1-null mice lacking voltage-gated Kv1.1 channels activate Fos expression in select limbic circuits.

Expression and function of Kv1.1 potassium channels in human atria from patients with atrial fibrillation.

Gradient Index Microlens Implanted in Prefrontal Cortex of Mouse Does Not Affect Behavioral Test Performance over Time.

Severe respiratory changes at end stage in a FUS-induced disease state in adult rats.

Scn2a deletion improves survival and brain-heart dynamics in the Kcna1-null mouse model of sudden unexpected death in epilepsy (SUDEP).

Pharmacogenetics of KCNQ channel activation in 2 potassium channelopathy mouse models of epilepsy.

Kv1.1 channel subunits in the control of neurocardiac function.

Neuron-specific Kv1.1 deficiency is sufficient to cause epilepsy, premature death, and cardiorespiratory dysregulation.

Clinical Spectrum of Mutations: New Insights into Episodic Ataxia and Epilepsy Comorbidity.

Decreased bioavailability of hydrogen sulfide links vascular endothelium and atrial remodeling in atrial fibrillation.

Kv1.1 potassium channel subunit deficiency alters ventricular arrhythmia susceptibility, contractility, and repolarization.

Kv1.1 deficiency alters repetitive and social behaviors in mice and rescues autistic-like behaviors due to Scn2a haploinsufficiency.

Kv1.1 subunits localize to cardiorespiratory brain networks in mice where their absence induces astrogliosis and microgliosis.