Baoling Liu

Synergistic effects of hypertonic saline and valproic acid in a lethal rat two-hit model.

Novel pharmacologic treatment attenuates septic shock and improves long-term survival.

Valproic acid for the treatment of hemorrhagic shock: a dose-optimization study.

Differential effects of fresh frozen plasma and normal saline on secondary brain damage in a large animal model of polytrauma, hemorrhage and traumatic brain injury.

Early treatment with lyophilized plasma protects the brain in a large animal model of combined traumatic brain injury and hemorrhagic shock.

Creating a prosurvival phenotype through a histone deacetylase inhibitor in a lethal two-hit model.

Selective inhibition of histone deacetylase 6 alters the composition of circulating blood cells in a lethal septic model.

Treatment with a histone deacetylase inhibitor, valproic acid, is associated with increased platelet activation in a large animal model of traumatic brain injury and hemorrhagic shock.

Selective histone deacetylase-6 inhibition attenuates stress responses and prevents immune organ atrophy in a lethal septic model.

Development of a novel neuroprotective strategy: combined treatment with hypothermia and valproic acid improves survival in hypoxic hippocampal cells.

Normal saline influences coagulation and endothelial function after traumatic brain injury and hemorrhagic shock in pigs.

Histone deacetylase inhibitor treatment attenuates coagulation imbalance in a lethal murine model of sepsis.

Citrullinated histone H3: a novel target for the treatment of sepsis.

Effect of pharmacologic resuscitation on the brain gene expression profiles in a swine model of traumatic brain injury and hemorrhage.

Histone deacetylase III as a potential therapeutic target for the treatment of lethal sepsis.

Entecavir promotes CD34⁺ stem cell proliferation in the peripheral blood and liver of chronic hepatitis B and liver cirrhosis patients.

Protective effect of suberoylanilide hydroxamic acid against lipopolysaccharide-induced liver damage in rodents.

Inhibition of histone deacetylase 6 improves long-term survival in a lethal septic model.

Hypothermia and valproic acid activate prosurvival pathways after hemorrhage.

Selective histone deacetylase 6 inhibition prolongs survival in a lethal two-hit model.

Early resuscitation with fresh frozen plasma for traumatic brain injury combined with hemorrhagic shock improves neurologic recovery.

Inhibition of peptidylarginine deiminase attenuates inflammation and improves survival in a rat model of hemorrhagic shock.

Inhibition of histone deacetylase 6 restores innate immune cells in the bone marrow in a lethal septic model.

Selective inhibition of histone deacetylase 6 promotes survival in a rat model of hemorrhagic shock.

Addition of low-dose valproic acid to saline resuscitation provides neuroprotection and improves long-term outcomes in a large animal model of combined traumatic brain injury and hemorrhagic shock.

Resuscitation with Valproic Acid Alters Inflammatory Genes in a Porcine Model of Combined Traumatic Brain Injury and Hemorrhagic Shock.

Inhibition of histone deacetylase 6 restores intestinal tight junction in hemorrhagic shock.

Alterations in the human proteome following administration of valproic acid.

Protective effect of Cl-amidine against CLP-induced lethal septic shock in mice.

Early resuscitation with lyophilized plasma provides equal neuroprotection compared with fresh frozen plasma in a large animal survival model of traumatic brain injury and hemorrhagic shock.

Valproic acid modulates platelet and coagulation function ex vivo.

Lung Protective Effects of Low-Volume Resuscitation and Pharmacologic Treatment of Swine Subjected to Polytrauma and Hemorrhagic Shock.

CitH3: a reliable blood biomarker for diagnosis and treatment of endotoxic shock.

Tubastatin A prevents hemorrhage-induced endothelial barrier dysfunction.

Histone deacetylase inhibitors: Isoform selectivity improves survival in a hemorrhagic shock model.

Inhibition of peptidylarginine deiminase alleviates LPS-induced pulmonary dysfunction and improves survival in a mouse model of lethal endotoxemia.

Protective Effect of Tubastatin A in CLP-Induced Lethal Sepsis.