Jae Gyun Oh

Pressure-overload cardiac hypertrophy is associated with distinct alternative splicing due to altered expression of splicing factors.

The role of SUMO-1 in cardiac oxidative stress and hypertrophy.

Increased stiffness is the major early abnormality in a pig model of severe aortic stenosis and predisposes to congestive heart failure in the absence of systolic dysfunction.

Small-molecule activation of SERCA2a SUMOylation for the treatment of heart failure.

Post-translational Modifications in Heart Failure: Small Changes, Big Impact.

Cardiac Stim1 Silencing Impairs Adaptive Hypertrophy and Promotes Heart Failure Through Inactivation of mTORC2/Akt Signaling.

Matricellular Protein CCN5 Reverses Established Cardiac Fibrosis.

A series of robust genetic indicators for definitive identification of cardiomyocytes.

A Decoy Peptide Targeted to Protein Phosphatase 1 Attenuates Degradation of SERCA2a in Vascular Smooth Muscle Cells.

Cardiac fibrosis and miR-433.

Tenascin-C in Cardiac Hypertrophy and Fibrosis: Friend or Foe?

Protein Phosphatase Inhibitor-1 Gene Therapy in a Swine Model of Nonischemic Heart Failure.

Experimental Models of Cardiovascular Diseases: Overview.

Measuring Cardiomyocyte Contractility and Calcium Handling In Vitro.

miR-146a Suppresses SUMO1 Expression and Induces Cardiac Dysfunction in Maladaptive Hypertrophy.

Experimental models of cardiac physiology and pathology.

Role of the PRC2-Six1-miR-25 signaling axis in heart failure.

Role of SIRT1 in Modulating Acetylation of the Sarco-Endoplasmic Reticulum Ca-ATPase in Heart Failure.

Cross-communication between fibroblasts and cardiomyocytes.

Cardioprotective role of APIP in myocardial infarction through ADORA2B.