Patrick T. Fueger

Transporter-mediated absorption is the primary route of entry and is required for passive absorption of intestinal glucose into the blood of conscious dogs.

Fiber type-specific determinants of Vmax for insulin-stimulated muscle glucose uptake in vivo.

Prior exercise enhances passive absorption of intraduodenal glucose.

Hexokinase II partial knockout impairs exercise-stimulated glucose uptake in oxidative muscles of mice.

Interaction of insulin and prior exercise in control of hepatic metabolism of a glucose load.

Distributed control of glucose uptake by working muscles of conscious mice: roles of transport and phosphorylation.

Hexokinase II overexpression improves exercise-stimulated but not insulin-stimulated muscle glucose uptake in high-fat-fed C57BL/6J mice.

AMP kinase-induced skeletal muscle glucose but not long-chain fatty acid uptake is dependent on nitric oxide.

AMPK stimulation increases LCFA but not glucose clearance in cardiac muscle in vivo.

Regulation of insulin-stimulated muscle glucose uptake in the conscious mouse: role of glucose transport is dependent on glucose phosphorylation capacity.

Heart-type fatty acid-binding protein reciprocally regulates glucose and fatty acid utilization during exercise.

Control of exercise-stimulated muscle glucose uptake by GLUT4 is dependent on glucose phosphorylation capacity in the conscious mouse.

Exercise-induced changes in insulin and glucagon are not required for enhanced hepatic glucose uptake after exercise but influence the fate of glucose within the liver.

Control of muscle glucose uptake: test of the rate-limiting step paradigm in conscious, unrestrained mice.

5-Aminoimidazole-4-carboxamide-1-beta-D-ribofuranoside causes acute hepatic insulin resistance in vivo.

Glucose phosphorylation as a barrier to muscle glucose uptake.

Hexokinase II protein content is a determinant of exercise endurance capacity in the mouse.

Mobilization of glucose from the liver during exercise and replenishment afterward.

Partial gene deletion of heart-type fatty acid-binding protein limits the severity of dietary-induced insulin resistance.

Pro- and antiapoptotic proteins regulate apoptosis but do not protect against cytokine-mediated cytotoxicity in rat islets and beta-cell lines.

Point-Counterpoint: Glucose phosphorylation is/is not a significant barrier to muscle glucose uptake by the working muscle.

Chronic treatment with sildenafil improves energy balance and insulin action in high fat-fed conscious mice.

Glucose kinetics and exercise tolerance in mice lacking the GLUT4 glucose transporter.

Phosphorylation barriers to skeletal and cardiac muscle glucose uptakes in high-fat fed mice: studies in mice with a 50% reduction of hexokinase II.

Galphaz negatively regulates insulin secretion and glucose clearance.

Trefoil factor 3 stimulates human and rodent pancreatic islet beta-cell replication with retention of function.

Stimulation of human and rat islet beta-cell proliferation with retention of function by the homeodomain transcription factor Nkx6.1.

Glucose metabolism in vivo in four commonly used inbred mouse strains.

Metabolic implications of reduced heart-type fatty acid binding protein in insulin resistant cardiac muscle.

Rap1 promotes multiple pancreatic islet cell functions and signals through mammalian target of rapamycin complex 1 to enhance proliferation.

The physiological regulation of glucose flux into muscle in vivo.

Cafeteria diet is a robust model of human metabolic syndrome with liver and adipose inflammation: comparison to high-fat diet.

PPAR-γ Activation Restores Pancreatic Islet SERCA2 Levels and Prevents β-Cell Dysfunction under Conditions of Hyperglycemic and Cytokine Stress.