Kimberly R. Byrnes

Quantitation of calcitonin gene-related peptide mRNA and neuronal cell death in facial motor nuclei following axotomy and 633 nm low power laser treatment.

Photobiomodulation improves cutaneous wound healing in an animal model of type II diabetes.

Light promotes regeneration and functional recovery and alters the immune response after spinal cord injury.

Low power laser irradiation alters gene expression of olfactory ensheathing cells in vitro.

Role of the cell cycle in the pathobiology of central nervous system trauma.

Expression of two temporally distinct microglia-related gene clusters after spinal cord injury.

Role of cell cycle proteins in CNS injury.

Cell cycle activation contributes to post-mitotic cell death and secondary damage after spinal cord injury.

Dual-confocal fiber-optic method for absolute measurement of refractive index and thickness of optically transparent media.

Roscovitine reduces neuronal loss, glial activation, and neurologic deficits after brain trauma.

Metabotropic glutamate receptor 5 activation inhibits microglial associated inflammation and neurotoxicity.

Metabotropic glutamate receptors as targets for multipotential treatment of neurological disorders.

Activation of metabotropic glutamate receptor 5 modulates microglial reactivity and neurotoxicity by inhibiting NADPH oxidase.

Cell cycle activation and CNS injury.

Activation of metabotropic glutamate receptor 5 improves recovery after spinal cord injury in rodents.

A combined scoring method to assess behavioral recovery after mouse spinal cord injury.

Role of microglia in neurotrauma.

Neuropathological differences between rats and mice after spinal cord injury.

Novel neuroinflammatory targets in the chronically injured spinal cord.

Selective CDK inhibitor limits neuroinflammation and progressive neurodegeneration after brain trauma.

Cyclin D1 gene ablation confers neuroprotection in traumatic brain injury.

Delayed inflammatory mRNA and protein expression after spinal cord injury.

Delayed mGluR5 activation limits neuroinflammation and neurodegeneration after traumatic brain injury.

Characterization of inflammatory gene expression and galectin-3 function after spinal cord injury in mice.