Fu-Shin Yu

Toll-like receptor 2-mediated expression of beta-defensin-2 in human corneal epithelial cells.

Toll-like receptor 3 agonist poly(I:C)-induced antiviral response in human corneal epithelial cells.

Herpes simplex virus 1 infection induces the expression of proinflammatory cytokines, interferons and TLR7 in human corneal epithelial cells.

Toll-like receptors and the eye.

Toll-like receptors and corneal innate immunity.

SRC-family tyrosine kinases in wound- and ligand-induced epidermal growth factor receptor activation in human corneal epithelial cells.

Lysophosphatidic acid promoting corneal epithelial wound healing by transactivation of epidermal growth factor receptor.

Staphylococcus aureus protein A induced inflammatory response in human corneal epithelial cells.

Wound-induced ATP release and EGF receptor activation in epithelial cells.

Cross talk between c-Met and epidermal growth factor receptor during retinal pigment epithelial wound healing.

Modulation of corneal epithelial innate immune response to pseudomonas infection by flagellin pretreatment.

Flagellin suppresses the inflammatory response and enhances bacterial clearance in a murine model of Pseudomonas aeruginosa keratitis.

Staphylococcus aureus lipoproteins trigger human corneal epithelial innate response through toll-like receptor-2.

Role of small GTPase Rho in regulating corneal epithelial wound healing.

Rho kinases regulate corneal epithelial wound healing.

ERK1/2 mediate wounding- and G-protein-coupled receptor ligands-induced EGFR activation via regulating ADAM17 and HB-EGF shedding.

Lack of MD-2 expression in human corneal epithelial cells is an underlying mechanism of lipopolysaccharide (LPS) unresponsiveness.

High glucose suppresses epidermal growth factor receptor/phosphatidylinositol 3-kinase/Akt signaling pathway and attenuates corneal epithelial wound healing.

Growth factors and corneal epithelial wound healing.

LL-37 via EGFR transactivation to promote high glucose-attenuated epithelial wound healing in organ-cultured corneas.

Normalization of wound healing and diabetic markers in organ cultured human diabetic corneas by adenoviral delivery of c-Met gene.

Toll-like receptor 2 ligand-induced protection against bacterial endophthalmitis.

Flagellin-induced corneal antimicrobial peptide production and wound repair involve a novel NF-kappaB-independent and EGFR-dependent pathway.

UBIAD1 mutation alters a mitochondrial prenyltransferase to cause Schnyder corneal dystrophy.

Topical flagellin protects the injured corneas from Pseudomonas aeruginosa infection.

Topical flagellin-mediated innate defense against Candida albicans keratitis.

Impaired epithelial wound healing and EGFR signaling pathways in the corneas of diabetic rats.

Corneal complications in streptozocin-induced type I diabetic rats.

Dendritic cell-epithelium interplay is a determinant factor for corneal epithelial wound repair.

Activation of JNK signaling mediates connective tissue growth factor expression and scar formation in corneal wound healing.

Histone deacetylases inhibitor Trichostatin A ameliorates DNFB-induced allergic contact dermatitis and reduces epidermal Langerhans cells in mice.

Reduced innervation and delayed re-innervation after epithelial wounding in type 2 diabetic Goto-Kakizaki rats.

Interferon regulatory factor-1 in flagellin-induced reprogramming: potential protective role of CXCL10 in cornea innate defense against Pseudomonas aeruginosa infection.

Genome-wide transcriptional analysis of differentially expressed genes in diabetic, healing corneal epithelial cells: hyperglycemia-suppressed TGFβ3 expression contributes to the delay of epithelial wound healing in diabetic corneas.

Targeting toll-like receptor signaling as a novel approach to prevent ocular infectious diseases.

Pathogenicity of ocular isolates of Acinetobacter baumannii in a mouse model of bacterial endophthalmitis.

Flagellin-induced expression of CXCL10 mediates direct fungal killing and recruitment of NK cells to the cornea in response to Candida albicans infection.

Matrix Metalloproteinase-13 as a Target for Suppressing Corneal Ulceration Caused by Pseudomonas aeruginosa Infection.

Hyperglycemia-suppressed expression of Serpine1 contributes to delayed epithelial wound healing in diabetic mouse corneas.

Chitinase 3-Like 1 Promotes Candida albicans Killing and Preserves Corneal Structure and Function by Controlling Host Antifungal Responses.

New insights into microRNAs in skin wound healing.

TGF-β1-induced transcription factor networks in Langerhans cell development and maintenance.

Dendritic cell dysfunction and diabetic sensory neuropathy in the cornea.

Targeting Imbalance between IL-1β and IL-1 Receptor Antagonist Ameliorates Delayed Epithelium Wound Healing in Diabetic Mouse Corneas.

TIM-4 is differentially expressed in the distinct subsets of dendritic cells in skin and skin-draining lymph nodes and controls skin Langerhans cell homeostasis.

Toll-like Receptor (TLR) Signaling Interacts with CREBH to Modulate High-density Lipoprotein (HDL) in Response to Bacterial Endotoxin.

Intraepithelial dendritic cells and sensory nerves are structurally associated and functional interdependent in the cornea.

Zika virus infects cells lining the blood-retinal barrier and causes chorioretinal atrophy in mouse eyes.

IL-24 Promotes Keratitis in C57BL/6 Mouse Corneas.

Sensory nerve regeneration after epithelium wounding in normal and diabetic cornea.

ISG15 in Host Defense Against Candida albicans Infection in a Mouse Model of Fungal Keratitis.

CXCL10 suppression of hem- and lymph-angiogenesis in inflamed corneas through MMP13.

Inhibition of Soluble Epoxide Hydrolase 2 Ameliorates Diabetic Keratopathy and Impaired Wound Healing in Mouse Corneas.

Opposing Effects of IL-1Ra and IL-36Ra on Innate Immune Response to Infection in C57BL/6 Mouse Corneas.

TSLP Protects Corneas From Pseudomonas aeruginosa Infection by Regulating Dendritic Cells and IL-23-IL-17 Pathway.

Opposing Effects of Neuropilin-1 and -2 on Sensory Nerve Regeneration in Wounded Corneas: Role of Sema3C in Ameliorating Diabetic Neurotrophic Keratopathy.

IL-17 Promotes Keratitis in C57BL/6 Mouse Corneas.

Role of VIP and Sonic Hedgehog Signaling Pathways in Mediating Epithelial Wound Healing, Sensory Nerve Regeneration, and Their Defects in Diabetic Corneas.

ISG15 Acts as a Mediator of Innate Immune Response to Pseudomonas aeruginosa Infection in C57BL/6J Mouse Corneas.

Role of IL-36γ/IL-36R Signaling in Corneal Innate Defense Against Candida albicans Keratitis.

IL-36α Enhances Host Defense against Keratitis in C57BL/6 Mouse Corneas.

The impact of sensory neuropathy and inflammation on epithelial wound healing in diabetic corneas.

Ocular nociception and neuropathic pain initiated by blue light stress in C57BL/6J mice.

Protective effects of resolvin D1 in Pseudomonas aeruginosa keratitis.

Diabetes Exacerbates Pseudomonas aeruginosa Keratitis in Streptozotocin-Induced and db/db Mice via Altering Programmed Cell Death Pathways.

Reply to Moulton et al.

Lack of Elevated Expression of TGFβ3 Contributes to the Delay of Epithelial Wound Healing in Diabetic Corneas.

Combination Nanomedicine Strategy for Preventing High-Risk Corneal Transplantation Rejection.