Elisabeth R. Barton

Muscle-specific expression of insulin-like growth factor I counters muscle decline in mdx mice.

Functional improvement of dystrophic muscle by myostatin blockade.

Correction of the dystrophic phenotype by in vivo targeting of muscle progenitor cells.

Viral expression of insulin-like growth factor-I enhances muscle hypertrophy in resistance-trained rats.

Rat supraspinatus muscle atrophy after tendon detachment.

Systemic administration of L-arginine benefits mdx skeletal muscle function.

Impact of sarcoglycan complex on mechanical signal transduction in murine skeletal muscle.

Viral expression of insulin-like growth factor-I isoforms promotes different responses in skeletal muscle.

The ABCs of IGF-I isoforms: impact on muscle hypertrophy and implications for repair.

PTC124 targets genetic disorders caused by nonsense mutations.

Sexual dimorphism of murine masticatory muscle function.

Genetic and pharmacologic inhibition of mitochondrial-dependent necrosis attenuates muscular dystrophy.

Expression profiling reveals heightened apoptosis and supports fiber size economy in the murine muscles of mastication.

The insulin-like growth factor (IGF)-I E-peptides modulate cell entry of the mature IGF-I protein.

Glucose regulated protein 94 is required for muscle differentiation through its control of the autocrine production of insulin-like growth factors.

The insulin-like growth factor (IGF)-I E-peptides are required for isoform-specific gene expression and muscle hypertrophy after local IGF-I production.

Systemic myostatin inhibition via liver-targeted gene transfer in normal and dystrophic mice.

Restoration of gamma-sarcoglycan localization and mechanical signal transduction are independent in murine skeletal muscle.

Comments on Point:Counterpoint: IGF is/is not the major physiological regulator of muscle mass. Is any factor necessary and sufficient for muscle mass regulation?

Overexpression of SERCA1a in the mdx diaphragm reduces susceptibility to contraction-induced damage.

Diaphragm displays early and progressive functional deficits in dysferlin-deficient mice.

Membrane blebbing as an assessment of functional rescue of dysferlin-deficient human myotubes via nonsense suppression.

Activin IIB receptor blockade attenuates dystrophic pathology in a mouse model of Duchenne muscular dystrophy.

Regulation of muscle mass by follistatin and activins.

IGF-I and vitamin C promote myogenic differentiation of mouse and human skeletal muscle cells at low temperatures.

The role of GH and IGF-I in mediating anabolic effects of testosterone on androgen-responsive muscle.

Chronic losartan administration reduces mortality and preserves cardiac but not skeletal muscle function in dystrophic mice.

Is functional hypertrophy and specific force coupled with the addition of myonuclei at the single muscle fiber level?

Rescue of dystrophic skeletal muscle by PGC-1α involves a fast to slow fiber type shift in the mdx mouse.

Deletion of muscle GRP94 impairs both muscle and body growth by inhibiting local IGF production.

HEXIM1 controls satellite cell expansion after injury to regulate skeletal muscle regeneration.

Insulin-like growth factor-I E-peptide activity is dependent on the IGF-I receptor.

Gene transfer of arginine kinase to skeletal muscle using adeno-associated virus.

Gamma-sarcoglycan is required for the response of archvillin to mechanical stimulation in skeletal muscle.

Masticatory muscles of mouse do not undergo atrophy in space.

Role of IGF-I signaling in muscle bone interactions.

Increased collagen cross-linking is a signature of dystrophin-deficient muscle.

Osteopontin ablation ameliorates muscular dystrophy by shifting macrophages to a pro-regenerative phenotype.

Activin Receptor Type IIB Inhibition Improves Muscle Phenotype and Function in a Mouse Model of Spinal Muscular Atrophy.

The IGF axis in HPV associated cancers.

Contrast-Enhanced Near-Infrared Optical Imaging Detects Exacerbation and Amelioration of Murine Muscular Dystrophy.

Regulation of fibrosis in muscular dystrophy.

Deletion of muscle IGF-I transiently impairs growth and progressively disrupts glucose homeostasis in male mice.

Generation and characterization of monoclonal antibodies that recognize human and murine supervillin protein isoforms.

Insulin-Like Growth Factor I Regulation and Its Actions in Skeletal Muscle.

Deleting nebulin's C-terminus reveals its importance to sarcomeric structure and function and is sufficient to invoke nemaline myopathy.

Smooth muscle atrophy and colon pathology in SMN deficient mice.

Functional muscle hypertrophy by increased insulin-like growth factor 1 does not require dysferlin.

A Key Role for the Ubiquitin Ligase UBR4 in Myofiber Hypertrophy in Drosophila and Mice.

Matrix Metalloproteinase 13 from Satellite Cells is Required for Efficient Muscle Growth and Regeneration.

The ties that bind: functional clusters in limb-girdle muscular dystrophy.

The D2.mdx mouse as a preclinical model of the skeletal muscle pathology associated with Duchenne muscular dystrophy.

Antagonistic control of myofiber size and muscle protein quality control by the ubiquitin ligase UBR4 during aging.

Hesperidin Promotes Osteogenesis and Modulates Collagen Matrix Organization and Mineralization In Vitro and In Vivo.

Actions and interactions of IGF-I and MMPs during muscle regeneration.

Muscle insulin-like growth factor-I modulates murine craniofacial bone growth.

Deletion of muscle exacerbates disuse atrophy weakness in mice.

The impact of hindlimb disuse on sepsis-induced myopathy in mice.

Pharmacologic approaches to prevent skeletal muscle atrophy after spinal cord injury.

Novel γ-sarcoglycan interactors in murine muscle membranes.

Isolated murine skeletal muscles utilize pyruvate over glucose for oxidation.

Modulating fast skeletal muscle contraction protects skeletal muscle in animal models of Duchenne muscular dystrophy.

Chloroplast transformation for bioencapsulation and oral delivery using the immunoglobulin G fragment crystallizable (Fc) domain.

Phosphorylation of AMPKα at Ser485/491 Is Dependent on Muscle Contraction and Not Muscle-Specific IGF-I Overexpression.

Chloroplast transformation for bioencapsulation and oral delivery using the immunoglobulin G fragment crystallizable (Fc) domain.

Loss of calpain 3 dysregulates store-operated calcium entry and its exercise response in mice.

Ablation of specific insulin-like growth factor I forms reveals the importance of cleavage for regenerative capacity and glycosylation for skeletal muscle storage.

Transcriptomics reveals transient and dynamic muscle fibrosis and atrophy differences following spinal cord injury in rats.