Matthew N. Rasband

Clustering of neuronal potassium channels is independent of their interaction with PSD-95.

Dysregulation of axonal sodium channel isoforms after adult-onset chronic demyelination.

Paranodal transverse bands are required for maintenance but not initiation of Nav1.6 sodium channel clustering in CNS optic nerve axons.

Integrin-linked kinase is required for laminin-2-induced oligodendrocyte cell spreading and CNS myelination.

The myelin-axolemmal complex: biochemical dissection and the role of galactosphingolipids.

Does paranode formation and maintenance require partitioning of neurofascin 155 into lipid rafts?

Proteomic mapping provides powerful insights into functional myelin biology.

It's "juxta" potassium channel!

BetaIV spectrins are essential for membrane stability and the molecular organization of nodes of Ranvier.

BetaIVSigma1 spectrin stabilizes the nodes of Ranvier and axon initial segments.

CNP is required for maintenance of axon-glia interactions at nodes of Ranvier in the CNS.

Where is the spike generator of the cochlear nerve? Voltage-gated sodium channels in the mouse cochlea.

Age-related molecular reorganization at the node of Ranvier.

Early events in node of Ranvier formation during myelination and remyelination in the PNS.

Spectrins and ankyrinB constitute a specialized paranodal cytoskeleton.

WAVE1 is required for oligodendrocyte morphogenesis and normal CNS myelination.

Intrinsic and extrinsic determinants of ion channel localization in neurons.

Glial regulation of the axonal membrane at nodes of Ranvier.

Neuron-glia interactions at the node of Ranvier.

betaIV spectrin is recruited to axon initial segments and nodes of Ranvier by ankyrinG.

alphaII-spectrin is essential for assembly of the nodes of Ranvier in myelinated axons.

Gangliosides contribute to stability of paranodal junctions and ion channel clusters in myelinated nerve fibers.

Anti-GM1 antibodies cause complement-mediated disruption of sodium channel clusters in peripheral motor nerve fibers.

A central role for Necl4 (SynCAM4) in Schwann cell-axon interaction and myelination.

Neurofascin assembles a specialized extracellular matrix at the axon initial segment.

Neurofascin as a novel target for autoantibody-mediated axonal injury.

Spectrin and ankyrin-based cytoskeletons at polarized domains in myelinated axons.

Postsynaptic density-93 clusters Kv1 channels at axon initial segments independently of Caspr2.

The functional organization and assembly of the axon initial segment.

Molecular mechanisms of node of Ranvier formation.

AnkyrinG is required for maintenance of the axon initial segment and neuronal polarity.

Electrical excitability of early neurons in the human cerebral cortex during the second trimester of gestation.

Na+ channels get anchored...with a little help.

Multiple molecular interactions determine the clustering of Caspr2 and Kv1 channels in myelinated axons.

Converging on the origins of axonal ion channel clustering.

Proteomic analysis of optic nerve lipid rafts reveals new paranodal proteins.

Disruption of the axon initial segment cytoskeleton is a new mechanism for neuronal injury.

ADAM22, a Kv1 channel-interacting protein, recruits membrane-associated guanylate kinases to juxtaparanodes of myelinated axons.

Novel forms of neurofascin 155 in the central nervous system: alterations in paranodal disruption models and multiple sclerosis.

The axon initial segment and the maintenance of neuronal polarity.

Clustered K+ channel complexes in axons.

A β(IV)-spectrin/CaMKII signaling complex is essential for membrane excitability in mice.

Composition, assembly, and maintenance of excitable membrane domains in myelinated axons.

Oligodendrocyte myelin glycoprotein does not influence node of ranvier structure or assembly.

Schwann cell spectrins modulate peripheral nerve myelination.

Maintenance of neuronal polarity.

Short- and long-term plasticity at the axon initial segment.

The axon initial segment in nervous system disease and injury.

Di-rectifying Tau.

Alterations in intrinsic membrane properties and the axon initial segment in a mouse model of Angelman syndrome.

Dysfunction of nodes of Ranvier: a mechanism for anti-ganglioside antibody-mediated neuropathies.

IκBα is not required for axon initial segment assembly.

An ankyrinG-binding motif is necessary and sufficient for targeting Nav1.6 sodium channels to axon initial segments and nodes of Ranvier.

A distal axonal cytoskeleton forms an intra-axonal boundary that controls axon initial segment assembly.

Galveston Brain Injury Conference 2010: clinical and experimental aspects of blast injury.

Blast wave exposure impairs memory and decreases axon initial segment length.

Neurofascin as a target for autoantibodies in peripheral neuropathies.

Remodeling of the axon initial segment after focal cortical and white matter stroke.

Cytoskeleton: axons earn their stripes.

Na+ channel-dependent recruitment of Navβ4 to axon initial segments and nodes of Ranvier.

Three mechanisms assemble central nervous system nodes of Ranvier.

Mechanisms of hearing loss after blast injury to the ear.

Genetic reduction of the α1 subunit of Na/K-ATPase corrects multiple hippocampal phenotypes in Angelman syndrome.

Excitable domains of myelinated nerves: axon initial segments and nodes of Ranvier.

Membrane domain organization of myelinated axons requires βII spectrin.

Computation identifies structural features that govern neuronal firing properties in slowly adapting touch receptors.