Suzanne M. Crowe

HIV-1 down-modulates gamma signaling chain of Fc gamma R in human macrophages: a possible mechanism for inhibition of phagocytosis.

Coreceptor phenotype of natural human immunodeficiency virus with nef deleted evolves in vivo, leading to increased virulence.

The role of monocytes and macrophages in the pathogenesis of HIV-1 infection.

Proline residues within spacer peptide p1 are important for human immunodeficiency virus type 1 infectivity, protein processing, and genomic RNA dimer stability.

Selectively reduced tat mRNA heralds the decline in productive human immunodeficiency virus type 1 infection in monocyte-derived macrophages.

Virion-associated cholesterol is critical for the maintenance of HIV-1 structure and infectivity.

The influence of cytokines, chemokines and their receptors on HIV-1 replication in monocytes and macrophages.

Analysis of HIV-1 viral load in seminal plasma samples.

Defective phagocytosis by human monocyte/macrophages following HIV-1 infection: underlying mechanisms and modulation by adjunctive cytokine therapy.

Immunological and virological failure after antiretroviral therapy is associated with enhanced peripheral and thymic pathogenicity.

HIV and cells of macrophage/dendritic lineage and other non-T cell reservoirs: new answers yield new questions.

Human immunodeficiency virus type 1 infection inhibits granulocyte-macrophage colony-stimulating factor-induced activation of STAT5A in human monocyte-derived macrophages.

Phagocytic efficiency of monocytes and macrophages obtained from Sydney blood bank cohort members infected with an attenuated strain of HIV-1.

The molecular epidemiology of HIV-1 in South and East Asia.

Interferon-gamma therapy activates human monocytes for enhanced phagocytosis of Mycobacterium avium complex in HIV-infected individuals.

Longitudinal analysis of nef/long terminal repeat-deleted HIV-1 in blood and cerebrospinal fluid of a long-term survivor who developed HIV-associated dementia.

Locking the door to HIV entry: how far can we go?

Pathogenesis of macrophage tropic HIV-1.

The molecular epidemiology of HIV type 1 among Vietnamese Australian injecting drug users in Melbourne, Australia.

Evaluation of a low cost reverse transcriptase assay for plasma HIV-1 viral load monitoring.

In vivo replication kinetics of a nef-deleted strain of HIV-1.

Uncoupling coreceptor usage of human immunodeficiency virus type 1 (HIV-1) from macrophage tropism reveals biological properties of CCR5-restricted HIV-1 isolates from patients with acquired immunodeficiency syndrome.

Antiretroviral compounds: mechanisms underlying failure of HAART to eradicate HIV-1.

Isolation of human immunodeficiency virus type 1 from peripheral blood monocytes.

Phenotypic characterization of blood monocytes from HIV-infected individuals.

Transendothelial migration of monocytes: the underlying molecular mechanisms and consequences of HIV-1 infection.

Phagocytosis stimulates mobilization and shedding of intracellular CD16A in human monocytes and macrophages: inhibition by HIV-1 infection.

Comparison of two human immunodeficiency virus (HIV) RNA surrogate assays to the standard HIV RNA assay.

Efficacy of Cyto-Chex blood preservative for delayed manual CD4 testing using Dynal T4 Quant CD4 test among HIV-infected persons in Zambia.

Normal CD16 expression and phagocytosis of Mycobacterium avium complex by monocytes from a current cohort of HIV-1-infected patients.

Adverse effects of antiretroviral drugs on HIV-1-infected and -uninfected human monocyte-derived macrophages.

Impaired complement-mediated phagocytosis by HIV type-1-infected human monocyte-derived macrophages involves a cAMP-dependent mechanism.

A reliable and inexpensive EasyCD4 assay for monitoring HIV-infected individuals in resource-limited settings.

Advances in macrophage and dendritic cell biology in HIV-1 infection stress key understudied areas in infection, pathogenesis, and analysis of viral reservoirs.

Matrix metalloproteinases, their production by monocytes and macrophages and their potential role in HIV-related diseases.

HIV-1 viral load assays for resource-limited settings.

The mechanism underlying defective Fcgamma receptor-mediated phagocytosis by HIV-1-infected human monocyte-derived macrophages.

Changes in the V3 region of gp120 contribute to unusually broad coreceptor usage of an HIV-1 isolate from a CCR5 Delta32 heterozygote.

The CD16+ monocyte subset is more permissive to infection and preferentially harbors HIV-1 in vivo.

CD16+ monocyte subset preferentially harbors HIV-1 and is expanded in pregnant Malawian women with Plasmodium falciparum malaria and HIV-1 infection.

The heterosexual HIV type 1 epidemic in Papua New Guinea is dominated by subtype C.

Pathogenicity and immunogenicity of attenuated, nef-deleted HIV-1 strains in vivo.

Use of an HIV-1 reverse-transcriptase enzyme-activity assay to measure HIV-1 viral load as a potential alternative to nucleic acid-based assay for monitoring antiretroviral therapy in resource-limited settings.

Reduction in hepatitis C-related liver disease associated with GB virus C in human immunodeficiency virus coinfection.

Kynurenine pathway metabolism in human blood-brain-barrier cells: implications for immune tolerance and neurotoxicity.

Effects of HIV-1 infection in vitro on transendothelial migration by monocytes and monocyte-derived macrophages.

Macrophages and residual HIV infection.

Enhanced monocyte Fc phagocytosis by a homologue of interleukin-10 encoded by human cytomegalovirus.

Evaluation of the Cavidi ExaVir Load assay (version 3) for plasma human immunodeficiency virus type 1 load monitoring.

Assessment of the low-cost Cavidi ExaVir Load assay for monitoring HIV viral load in pediatric and adult patients.

Evaluation of the blood stabilizers TransFix and Cyto-Chex BCT for low-cost CD4 T-cell methodologies.

Effect of reverse transcriptase inhibitors and mutations on the low-cost Cavidi reverse transcriptase viral load assay.

An HIV-1 integrase genotype assay for the detection of drug resistance mutations.

The macrophage: the intersection between HIV infection and atherosclerosis.

A novel, rapid method to detect infectious HIV-1 from plasma of persons infected with HIV-1.

Stability of dried blood spots for HIV-1 drug resistance analysis.

Decreased NK Cell FcRgamma in HIV-1 infected individuals receiving combination antiretroviral therapy: a cross sectional study.

Quantifying HIV for monitoring antiretroviral therapy in resource-poor settings.

Editorial: Monocyte subpopulations and lentiviral infection.

Extremely prolonged HIV seroconversion associated with an MHC haplotype carrying disease susceptibility genes for antibody deficiency disorders.

HIV reverse transcriptase activity assay: a feasible surrogate for HIV viral load measurement in China.

HIV inhibits early signal transduction events triggered by CD16 cross-linking on NK cells, which are important for antibody-dependent cellular cytotoxicity.

Both CD31(+) and CD31⁻ naive CD4(+) T cells are persistent HIV type 1-infected reservoirs in individuals receiving antiretroviral therapy.

Point-of-care testing.

Biomarkers of immune dysfunction following combination antiretroviral therapy for HIV infection.

Symptomatic and asymptomatic early neurosyphilis in HIV-infected men who have sex with men: a retrospective case series from 2000 to 2007.

Differential expression of CD163 on monocyte subsets in healthy and HIV-1 infected individuals.

Antiviral activity of arbidol, a broad-spectrum drug for use against respiratory viruses, varies according to test conditions.

HIV infection and aging of the innate immune system.

HIV infection induces age-related changes to monocytes and innate immune activation in young men that persist despite combination antiretroviral therapy.

Endothelial cell activation promotes foam cell formation by monocytes following transendothelial migration in an in vitro model.

Aging is associated with chronic innate immune activation and dysregulation of monocyte phenotype and function.

Virologically suppressed HIV patients show activation of NK cells and persistent innate immune activation.

Evaluating new CD4 enumeration technologies for resource-constrained countries.

CRF01_AE dominates the HIV-1 epidemic in Indonesia.

Are monocytes the canary in the coal mine for HIV-related atherosclerosis?

Inhibition of telomerase activity by human immunodeficiency virus (HIV) nucleos(t)ide reverse transcriptase inhibitors: a potential factor contributing to HIV-associated accelerated aging.

Age-associated changes in monocyte and innate immune activation markers occur more rapidly in HIV infected women.

First molecular epidemiology study of Mycobacterium tuberculosis in Kiribati.

Characterization of tetraspanins CD9, CD53, CD63, and CD81 in monocytes and macrophages in HIV-1 infection.

HIV epidemic in men who have sex with men in Philippines.

Associations between surface markers on blood monocytes and carotid atherosclerosis in HIV-positive individuals.

Increased glucose metabolic activity is associated with CD4+ T-cell activation and depletion during chronic HIV infection.

Acquisition of HIV by African-born residents of Victoria, Australia: insights from molecular epidemiology.

Inflammatory co-morbidities in HIV+ individuals: learning lessons from healthy ageing.

Systematic review of the performance of HIV viral load technologies on plasma samples.

Systematic review of the use of dried blood spots for monitoring HIV viral load and for early infant diagnosis.

How does monocyte metabolism impact inflammation and aging during chronic HIV infection?

Presence of HIV drug resistance in antiretroviral therapy-naive and -experienced patients from Papua New Guinea.

Monocytes as regulators of inflammation and HIV-related comorbidities during cART.

The importance of monocytes and macrophages in HIV pathogenesis, treatment, and cure.

Tuberculosis (TB)-associated immune reconstitution inflammatory syndrome in TB-HIV co-infected patients in Malaysia: prevalence, risk factors, and treatment outcomes.

Macrophage migration inhibitory factor: a potential biomarker for cardiovascular disease in persons with HIV?

Glucose transporter 1-expressing proinflammatory monocytes are elevated in combination antiretroviral therapy-treated and untreated HIV+ subjects.

C-reactive protein as a predictor of cardiovascular risk in HIV-infected individuals.

Plasma interleukin-18 levels are a biomarker of innate immune responses that predict and characterize tuberculosis-associated immune reconstitution inflammatory syndrome.

Viremic and Virologically Suppressed HIV Infection Increases Age-Related Changes to Monocyte Activation Equivalent to 12 and 4 Years of Aging, Respectively.

Glucose metabolism regulates T cell activation, differentiation, and functions.

CD4 enumeration technologies: a systematic review of test performance for determining eligibility for antiretroviral therapy.

The dire sexual health crisis among MSM in the Philippines: an exploding HIV epidemic in the absence of essential health services.

Quantitative Assessment of Intra-Patient Variation in CD4+ T Cell Counts in Stable, Virologically-Suppressed, HIV-Infected Subjects.

Monocytes from HIV-infected individuals show impaired cholesterol efflux and increased foam cell formation after transendothelial migration.

Factors Influencing Antiretroviral Adherence and Virological Outcomes in People Living with HIV in the Highlands of Papua New Guinea.

Planning for the Next Global Pandemic.

Panobinostat clinical trial highlights the challenges towards an HIV cure.

Regulators of glucose metabolism in CD4(+) and CD8(+) T cells.

Aberrant Inflammasome Activation Characterizes Tuberculosis-Associated Immune Reconstitution Inflammatory Syndrome.

Emerging Role and Characterization of Immunometabolism: Relevance to HIV Pathogenesis, Serious Non-AIDS Events, and a Cure.

Glucose Metabolism in T Cells and Monocytes: New Perspectives in HIV Pathogenesis.

Acceptability and feasibility of point-of-care CD4 testing on HIV continuum of care in low and middle income countries: a systematic review.

Genetic polymorphisms in the CD14 gene are associated with monocyte activation and carotid intima-media thickness in HIV-infected patients on antiretroviral therapy.