Reuben Kim

The telomeric length and heterogeneity decrease with age in normal human oral keratinocytes.

Senescence-associated decline in the intranuclear accumulation of hOGG1-alpha and impaired 8-oxo-dG repair activity in senescing normal human oral keratinocytes in vivo.

Abnormal DNA end-joining activity in human head and neck cancer.

Heterogeneous nuclear ribonucleoprotein G shows tumor suppressive effect against oral squamous cell carcinoma cells.

Bmi-1 cooperates with human papillomavirus type 16 E6 to immortalize normal human oral keratinocytes.

p53 promotes the fidelity of DNA end-joining activity by, in part, enhancing the expression of heterogeneous nuclear ribonucleoprotein G.

hnRNP G elicits tumor-suppressive activity in part by upregulating the expression of Txnip.

HIV-1 Tat enhances replicative potential of human oral keratinocytes harboring HPV-16 genome.

Association of hsp90 to the hTERT promoter is necessary for hTERT expression in human oral cancer cells.

Bmi-1 extends the life span of normal human oral keratinocytes by inhibiting the TGF-beta signaling.

Grainyhead-like 2 enhances the human telomerase reverse transcriptase gene expression by inhibiting DNA methylation at the 5'-CpG island in normal human keratinocytes.

miR-181a shows tumor suppressive effect against oral squamous cell carcinoma cells by downregulating K-ras.

Radioprotective effects of Bmi-1 involve epigenetic silencing of oxidase genes and enhanced DNA repair in normal human keratinocytes.

Impaired odontogenic differentiation of senescent dental mesenchymal stem cells is associated with loss of Bmi-1 expression.

Identification of senescence-inducing microRNAs in normal human keratinocytes.

Expression and mutation analysis of heterogeneous nuclear ribonucleoprotein G in human oral cancer.

DeltaNp63α protein triggers epithelial-mesenchymal transition and confers stem cell properties in normal human keratinocytes.

TNFα enhances cancer stem cell-like phenotype via Notch-Hes1 activation in oral squamous cell carcinoma cells.

Therapeutic potential of mesenchymal stem cells for oral and systemic diseases.

Camphorquinone inhibits odontogenic differentiation of dental pulp cells and triggers release of inflammatory cytokines.

Efavirenz induces autophagy and aberrant differentiation in normal human keratinocytes.

2-Hydroxyethyl methacrylate inhibits migration of dental pulp stem cells.

A polymorphism in IRF4 affects human pigmentation through a tyrosinase-dependent MITF/TFAP2A pathway.

Development of oral osteomucosal tissue constructs in vitro and localization of fluorescently-labeled bisphosphonates to hard and soft tissue.

Gene expression signatures affected by ethanol and/or nicotine in normal human normal oral keratinocytes (NHOKs).

Impaired bone resorption and woven bone formation are associated with development of osteonecrosis of the jaw-like lesions by bisphosphonate and anti-receptor activator of NF-κB ligand antibody in mice.

Osteo-/odontogenic differentiation of induced mesenchymal stem cells generated through epithelial-mesenchyme transition of cultured human keratinocytes.

The p63 Gene Is Regulated by Grainyhead-like 2 (GRHL2) through Reciprocal Feedback and Determines the Epithelial Phenotype in Human Keratinocytes.

Regulation of p53 during senescence in normal human keratinocytes.

Elevated expression of JMJD6 is associated with oral carcinogenesis and maintains cancer stemness properties.

Human papillomavirus 16 (HPV16) enhances tumor growth and cancer stemness of HPV-negative oral/oropharyngeal squamous cell carcinoma cells via miR-181 regulation.

Managing Intraoral Lesions in Oral Cancer Patients in a General Dental Practice: An Overview.

Grainyhead-like 2 regulates epithelial plasticity and stemness in oral cancer cells.

Modeling the Etiology of p53-mutated Cancer Cells.

Orai1 mediates osteogenic differentiation via BMP signaling pathway in bone marrow mesenchymal stem cells.

Orai1 promotes tumor progression by enhancing cancer stemness via NFAT signaling in oral/oropharyngeal squamous cell carcinoma.

IL-36 Induces Bisphosphonate-Related Osteonecrosis of the Jaw-Like Lesions in Mice by Inhibiting TGF-β-Mediated Collagen Expression.

Preexisting Periapical Inflammatory Condition Exacerbates Tooth Extraction-induced Bisphosphonate-related Osteonecrosis of the Jaw Lesions in Mice.

Clinical and Molecular Perspectives of Reparative Dentin Formation: Lessons Learned from Pulp-Capping Materials and the Emerging Roles of Calcium.