Judith Berman

MEC3, MEC1, and DDC2 are essential components of a telomere checkpoint pathway required for cell cycle arrest during senescence in Saccharomyces cerevisiae.

A forkhead transcription factor is important for true hyphal as well as yeast morphogenesis in Candida albicans.

Candida Albicans: a molecular revolution built on lessons from budding yeast.

mRNAs encoding telomerase components and regulators are controlled by UPF genes in Saccharomyces cerevisiae.

The effects of dehydration on rehabilitation outcomes of elderly orthopedic patients.

Molecular genetic and genomic approaches to the study of medically important fungi.

Functional conservation of Dhh1p, a cytoplasmic DExD/H-box protein present in large complexes.

Telomere cap components influence the rate of senescence in telomerase-deficient yeast cells.

Cassettes for the PCR-mediated construction of regulatable alleles in Candida albicans.

The distinct morphogenic states of Candida albicans.

Yeast chromatin assembly complex 1 protein excludes nonacetylatable forms of histone H4 from chromatin and the nucleus.

Transcriptional profiling in Candida albicans reveals new adaptive responses to extracellular pH and functions for Rim101p.

Comparative genome hybridization reveals widespread aneuploidy in Candida albicans laboratory strains.

The mitotic cyclins Clb2p and Clb4p affect morphogenesis in Candida albicans.

Candida albicans hyphae have a Spitzenkörper that is distinct from the polarisome found in yeast and pseudohyphae.

Rewiring of the yeast transcriptional network through the evolution of motif usage.

A human-curated annotation of the Candida albicans genome.

Microtubules in Candida albicans hyphae drive nuclear dynamics and connect cell cycle progression to morphogenesis.

A mutation in Tac1p, a transcription factor regulating CDR1 and CDR2, is coupled with loss of heterozygosity at chromosome 5 to mediate antifungal resistance in Candida albicans.

Comparative gene expression analysis by differential clustering approach: application to the Candida albicans transcription program.

Aneuploidy and isochromosome formation in drug-resistant Candida albicans.

Transcript profiles of Candida albicans cortical actin patch mutants reflect their cellular defects: contribution of the Hog1p and Mkc1p signaling pathways.

Morphogenesis and cell cycle progression in Candida albicans.

The pattern and evolution of yeast promoter bendability.

Genotypic evolution of azole resistance mechanisms in sequential Candida albicans isolates.

Haplotype mapping of a diploid non-meiotic organism using existing and induced aneuploidies.

Dynein-dependent nuclear dynamics affect morphogenesis in Candida albicans by means of the Bub2p spindle checkpoint.

An isochromosome confers drug resistance in vivo by amplification of two genes, ERG11 and TAC1.

The parasexual cycle in Candida albicans provides an alternative pathway to meiosis for the formation of recombinant strains.

Molecular architecture of the kinetochore-microtubule attachment site is conserved between point and regional centromeres.

Neocentromeres form efficiently at multiple possible loci in Candida albicans.

Evolution in Candida albicans populations during a single passage through a mouse host.

Evolution of pathogenicity and sexual reproduction in eight Candida genomes.

Additional cassettes for epitope and fluorescent fusion proteins in Candida albicans.

Efficient and rapid identification of Candida albicans allelic status using SNP-RFLP.

Aneuploid chromosomes are highly unstable during DNA transformation of Candida albicans.

SLA2 mutations cause SWE1-mediated cell cycle phenotypes in Candida albicans and Saccharomyces cerevisiae.

Acquisition of aneuploidy provides increased fitness during the evolution of antifungal drug resistance.

Growth and development: eukaryotes.

Dancing genomes: fungal nuclear positioning.

Genomic plasticity of the human fungal pathogen Candida albicans.

Low dosage of histone H4 leads to growth defects and morphological changes in Candida albicans.

Epigenetically-inherited centromere and neocentromere DNA replicates earliest in S-phase.

Evolutionary genomics: When abnormality is beneficial.

CaMtw1, a member of the evolutionarily conserved Mis12 kinetochore protein family, is required for efficient inner kinetochore assembly in the pathogenic yeast Candida albicans.

The requirement for the Dam1 complex is dependent upon the number of kinetochore proteins and microtubules.

Skin-resident murine dendritic cell subsets promote distinct and opposing antigen-specific T helper cell responses.

Evolutionary dynamics of Candida albicans during in vitro evolution.

Does stress induce (para)sex? Implications for Candida albicans evolution.

High-Resolution SNP/CGH Microarrays Reveal the Accumulation of Loss of Heterozygosity in Commonly Used Candida albicans Strains.

Flexibility of centromere and kinetochore structures.

Neocentromeres and epigenetically inherited features of centromeres.

Analysis of protein function in clinical C. albicans isolates.

Cell-cycle-coupled structural oscillation of centromeric nucleosomes in yeast.

Candida albicans.

The three clades of the telomere-associated TLO gene family of Candida albicans have different splicing, localization, and expression features.

Shuttle vectors for facile gap repair cloning and integration into a neutral locus in Candida albicans.

The 'obligate diploid' Candida albicans forms mating-competent haploids.

Monopolin recruits condensin to organize centromere DNA and repetitive DNA sequences.

A tetraploid intermediate precedes aneuploid formation in yeasts exposed to fluconazole.

Silencing is noisy: population and cell level noise in telomere-adjacent genes is dependent on telomere position and sir2.

Rapid mechanisms for generating genome diversity: whole ploidy shifts, aneuploidy, and loss of heterozygosity.

Origin replication complex binding, nucleosome depletion patterns, and a primary sequence motif can predict origins of replication in a genome with epigenetic centromeres.

Telomeric ORFs (TLOs) in Candida spp. Encode mediator subunits that regulate distinct virulence traits.

Genetic and phenotypic intra-species variation in Candida albicans.

YMAP: a pipeline for visualization of copy number variation and loss of heterozygosity in eukaryotic pathogens.

The evolution of drug resistance in clinical isolates of Candida albicans.

Telomeric ORFS in Candida albicans: does mediator tail wag the yeast?

Targeting the adaptability of heterogeneous aneuploids.

Candida albicans morphology and dendritic cell subsets determine T helper cell differentiation.

Real-Time Evolution of a Subtelomeric Gene Family in Candida albicans.

Parasexual Ploidy Reduction Drives Population Heterogeneity Through Random and Transient Aneuploidy in Candida albicans.

Shift and adapt: the costs and benefits of karyotype variations.

Physical limits on kinesin-5-mediated chromosome congression in the smallest mitotic spindles.

Polyploid titan cells produce haploid and aneuploid progeny to promote stress adaptation.

Corrigendum: The 'obligate diploid' Candida albicans forms mating-competent haploids.

High frame-rate resolution of cell division during Candida albicans filamentation.

Ploidy plasticity: a rapid and reversible strategy for adaptation to stress.

Candida albicans repetitive elements display epigenetic diversity and plasticity.

diskImageR: quantification of resistance and tolerance to antimicrobial drugs using disk diffusion assays.

Phenotypic Consequences of a Spontaneous Loss of Heterozygosity in a Common Laboratory Strain of Candida albicans.

Sir2 regulates stability of repetitive domains differentially in the human fungal pathogen Candida albicans.

Heteroresistance to Fluconazole Is a Continuously Distributed Phenotype among Candida glabrata Clinical Strains Associated with In Vivo Persistence.

Neocentromeres Provide Chromosome Segregation Accuracy and Centromere Clustering to Multiple Loci along a Candida albicans Chromosome.

Ploidy dynamics and evolvability in fungi.

Multidrug-Resistant Candida haemulonii and C. auris, Tel Aviv, Israel.

Haplotyping a Non-meiotic Diploid Fungal Pathogen Using Induced Aneuploidies and SNP/CGH Microarray Analysis.

Ploidy tug-of-war: evolutionary and genetic environments influence the rate of ploidy drive in a human fungal pathogen.