Oleksandr Platoshyn

Nitric oxide induces apoptosis by activating K+ channels in pulmonary vascular smooth muscle cells.

Activation of K(+) channels and increased migration of differentiated intestinal epithelial cells after wounding.

Role of capacitative Ca2+ entry in bronchial contraction and remodeling.

Inhibition of endogenous TRP1 decreases capacitative Ca2+ entry and attenuates pulmonary artery smooth muscle cell proliferation.

Cytochrome c activates K+ channels before inducing apoptosis.

PDGF stimulates pulmonary vascular smooth muscle cell proliferation by upregulating TRPC6 expression.

Apoptosis repressor with caspase domain inhibits cardiomyocyte apoptosis by reducing K+ currents.

Bone morphogenetic proteins induce apoptosis in human pulmonary vascular smooth muscle cells.

Hypoxia increases AP-1 binding activity by enhancing capacitative Ca2+ entry in human pulmonary artery endothelial cells.

Diversity of voltage-dependent K+ channels in human pulmonary artery smooth muscle cells.

Overexpression of human KCNA5 increases IK V and enhances apoptosis.

Overexpression of TRPC1 enhances pulmonary vasoconstriction induced by capacitative Ca2+ entry.

Enhanced expression of transient receptor potential channels in idiopathic pulmonary arterial hypertension.

Postnatal isl1+ cardioblasts enter fully differentiated cardiomyocyte lineages.

Identification of functional voltage-gated Na(+) channels in cultured human pulmonary artery smooth muscle cells.

Acute hypoxia selectively inhibits KCNA5 channels in pulmonary artery smooth muscle cells.

Long QT syndrome-associated I593R mutation in HERG potassium channel activates ER stress pathways.

TRPC1 functions as a store-operated Ca2+ channel in intestinal epithelial cells and regulates early mucosal restitution after wounding.

Divergent effects of BMP-2 on gene expression in pulmonary artery smooth muscle cells from normal subjects and patients with idiopathic pulmonary arterial hypertension.

Bone morphogenetic protein-2 upregulates expression and function of voltage-gated K+ channels in human pulmonary artery smooth muscle cells.

Thiol oxidation causes pulmonary vasodilation by activating K+ channels and inhibiting store-operated Ca2+ channels.

Function of Kv1.5 channels and genetic variations of KCNA5 in patients with idiopathic pulmonary arterial hypertension.

Dynamics of alpha-synuclein aggregation and inhibition of pore-like oligomer development by beta-synuclein.

Cystic fibrosis transmembrane conductance regulator (CFTR) functionality is dependent on coatomer protein I (COPI).

Heterogeneity of hypoxia-mediated decrease in I(K(V)) and increase in [Ca2+](cyt) in pulmonary artery smooth muscle cells.

Functional characterization of voltage-gated K+ channels in mouse pulmonary artery smooth muscle cells.

Hypoxia divergently regulates production of reactive oxygen species in human pulmonary and coronary artery smooth muscle cells.

Characterization of agonist-induced vasoconstriction in mouse pulmonary artery.

Downregulation of connexin40 is associated with coronary endothelial cell dysfunction in streptozotocin-induced diabetic mice.

p75 neurotrophin receptor regulates agonist-induced pulmonary vasoconstriction.

Mechanisms of hybrid oligomer formation in the pathogenesis of combined Alzheimer's and Parkinson's diseases.

Hypoxia selectively inhibits KCNA5 channels in pulmonary artery smooth muscle cells.

Tetramerization domain mutations in KCNA5 affect channel kinetics and cause abnormal trafficking patterns.

Functional ion channels in human pulmonary artery smooth muscle cells: Voltage-dependent cation channels.

Combinational spinal GAD65 gene delivery and systemic GABA-mimetic treatment for modulation of spasticity.

ALS-linked TDP-43 mutations produce aberrant RNA splicing and adult-onset motor neuron disease without aggregation or loss of nuclear TDP-43.

Amelioration of motor/sensory dysfunction and spasticity in a rat model of acute lumbar spinal cord injury by human neural stem cell transplantation.

Vinculin directly binds zonula occludens-1 and is essential for stabilizing connexin-43-containing gap junctions in cardiac myocytes.

Thoracic 9 Spinal Transection-Induced Model of Muscle Spasticity in the Rat: A Systematic Electrophysiological and Histopathological Characterization.

Gain of Toxicity from ALS/FTD-Linked Repeat Expansions in C9ORF72 Is Alleviated by Antisense Oligonucleotides Targeting GGGGCC-Containing RNAs.