Julian H. Lombard

Microvascular flow and tissue PO(2) in skeletal muscle of chronic reduced renal mass hypertensive rats.

Parenchymal tissue cytochrome P450 4A enzymes contribute to oxygen-induced alterations in skeletal muscle arteriolar tone.

High-salt diet depresses acetylcholine reactivity proximal to NOS activation in cerebral arteries.

Integration of hypoxic dilation signaling pathways for skeletal muscle resistance arteries.

Foreword to the special issue on microcirculatory adaptations to hypertension.

Interaction of myogenic mechanisms and hypoxic dilation in rat middle cerebral arteries.

High-salt diet impairs vascular relaxation mechanisms in rat middle cerebral arteries.

Skeletal muscle arteriolar reactivity in SS.BN13 consomic rats and Dahl salt-sensitive rats.

Chronic intermittent hypoxia impairs endothelium-dependent dilation in rat cerebral and skeletal muscle resistance arteries.

Effect of high-salt diet on NO release and superoxide production in rat aorta.

Introgression of chromosome 13 in Dahl salt-sensitive genetic background restores cerebral vascular relaxation.

Chronic AT(1) receptor blockade alters mechanisms mediating responses to hypoxia in rat skeletal muscle resistance arteries.

Evaluation of cytochrome P450-4A omega-hydroxylase and 20-hydroxyeicosatetraenoic acid as an O2 sensing mechanism in the microcirculation.

Expression of cytochrome P450-4A isoforms in the rat cremaster muscle microcirculation.

Salt-induced ANG II suppression impairs the response of cerebral artery smooth muscle cells to prostacyclin.

Effects of high-salt diet on CYP450-4A omega-hydroxylase expression and active tone in mesenteric resistance arteries.

Reduced angiotensin II and oxidative stress contribute to impaired vasodilation in Dahl salt-sensitive rats on low-salt diet.

Arteriolar responses to vasodilator stimuli and elevated P(O2) in renin congenic and Dahl salt-sensitive rats.

Restoration of normal vascular relaxation mechanisms in cerebral arteries by chromosomal substitution in consomic SS.13BN rats.

Chronic At1 receptor blockade alters the mechanisms mediating hypoxic dilation in middle cerebral arteries.

Chronic intermittent hypoxia alters NE reactivity and mechanics of skeletal muscle resistance arteries.

BN phenome: detailed characterization of the cardiovascular, renal, and pulmonary systems of the sequenced rat.

Role of superoxide and angiotensin II suppression in salt-induced changes in endothelial Ca2+ signaling and NO production in rat aorta.

Effect of high-salt diet on vascular relaxation and oxidative stress in mesenteric resistance arteries.

Modulation of vascular O2 responses by cytochrome 450-4A omega-hydroxylase metabolites in Dahl salt-sensitive rats.

Time-course and mechanisms of restored vascular relaxation by reduced salt intake and angiotensin II infusion in rats fed a high-salt diet.

Reduced oxidant stress, increased NO-dependent vasodilatation, and improved endothelial function with voluntary exercise in old mice: another excuse for long walks on the beach.

Angiotensin II maintains cerebral vascular relaxation via EGF receptor transactivation and ERK1/2.

CYP450 4A inhibition attenuates O2 induced arteriolar constriction in chronic but not acute Goldblatt hypertension.

Restoration of cerebral vascular relaxation in renin congenic rats by introgression of the Dahl R renin gene.

Uncoupling protein 2 (UCP2): Another player in the complex drama of vascular salt sensitivity.

Angiotensin-(1-7) and low-dose angiotensin II infusion reverse salt-induced endothelial dysfunction via different mechanisms in rat middle cerebral arteries.

Vascular responses in aortic rings of a consomic rat panel derived from the Fawn Hooded Hypertensive strain.

Impaired relaxation of cerebral arteries in the absence of elevated salt intake in normotensive congenic rats carrying the Dahl salt-sensitive renin gene.

Modulation by cytochrome P450-4A ω-hydroxylase enzymes of adrenergic vasoconstriction and response to reduced PO₂ in mesenteric resistance arteries of Dahl salt-sensitive rats.

Introgression of the Brown Norway renin allele onto the Dahl salt-sensitive genetic background increases Cu/Zn SOD expression in cerebral arteries.

Acute and chronic angiotensin-(1-7) restores vasodilation and reduces oxidative stress in mesenteric arteries of salt-fed rats.

Dahl salt-sensitive rats are protected against vascular defects related to diet-induced obesity.

Introgression of Brown Norway CYP4A genes on to the Dahl salt-sensitive background restores vascular function in SS-5(BN) consomic rats.

PPAR-ϒ pathway to vascular dysfunction.

Role of the CYP4A/20-HETE pathway in vascular dysfunction of the Dahl salt-sensitive rat.

Low-dose angiotensin II infusion restores vascular function in cerebral arteries of high salt-fed rats by increasing copper/zinc superoxide dimutase expression.

Deficiency of renal cortical EGF increases ENaC activity and contributes to salt-sensitive hypertension.

Reduced angiotensin II levels cause generalized vascular dysfunction via oxidant stress in hamster cheek pouch arterioles.

AT1 receptors prevent salt-induced vascular dysfunction in isolated middle cerebral arteries of 2 kidney-1 clip hypertensive rats.

FMRI and fcMRI phenotypes map the genomic effect of chromosome 13 in Brown Norway and Dahl salt-sensitive rats.

Amelioration of salt-induced vascular dysfunction in mesenteric arteries of Dahl salt-sensitive rats by missense mutation of extracellular superoxide dismutase.

Response to "Does angiotensin-dependent superoxide production help to prevent salt-induced endothelial dysfunction in 2 kidney-1 clip hypertensive rats?".

Mutation of Plekha7 attenuates salt-sensitive hypertension in the rat.

Vascular dysfunction precedes hypertension associated with a blood pressure locus on rat chromosome 12.

Enhancement of resting-state fcMRI networks by prior sensory stimulation.

The role of cyclo-oxygenase-1 in high-salt diet-induced microvascular dysfunction in humans.

The NRF2 knockout rat: a new animal model to study endothelial dysfunction, oxidant stress, and microvascular rarefaction.

Increased peripheral vascular disease risk progressively constrains perfusion adaptability in the skeletal muscle microcirculation.

Salt, Angiotensin II, Superoxide, and Endothelial Function.

Role of vascular reactive oxygen species in regulating cytochrome P450-4A enzyme expression in Dahl salt-sensitive rats.

Angiotensin-(1-7) Selectively Induces Relaxation and Modulates Endothelium-Dependent Dilation in Mesenteric Arteries of Salt-Fed Rats.

Mechanisms of Mas1 Receptor-Mediated Signaling in the Vascular Endothelium.

Contribution of mitochondria-derived free radicals to endothelial dysfunction in human skeletal muscle feed arteries: another hazard of the ageing process.

High salt intake shifts the mechanisms of flow-induced dilation in the middle cerebral arteries of Sprague-Dawley rats.

Region-Based Convolutional Neural Nets for Localization of Glomeruli in Trichrome-Stained Whole Kidney Sections.

Do computers dream of electric glomeruli?

High salt diet impairs cerebral blood flow regulation via salt-induced angiotensin II suppression.

NRF2 activation with Protandim attenuates salt-induced vascular dysfunction and microvascular rarefaction.