Cecilia Williams

The mutagenic effect of ultraviolet-A1 on human skin demonstrated by sequencing the p53 gene in single keratinocytes.

Catalog of gene expression in adult neural stem cells and their in vivo microenvironment.

Global gene expression analyses of hematopoietic stem cell-like cell lines with inducible Lhx2 expression.

Gene array identification of Ipf1/Pdx1-/- regulated genes in pancreatic progenitor cells.

Gene expression in murine mammary epithelial stem cell-like cells shows similarities to human breast cancer gene expression.

Tumor repressive functions of estrogen receptor beta in SW480 colon cancer cells.

Estrogen receptor β induces antiinflammatory and antitumorigenic networks in colon cancer cells.

The two-pore domain potassium channel KCNK5: induction by estrogen receptor alpha and role in proliferation of breast cancer cells.

Estradiol-activated estrogen receptor α does not regulate mature microRNAs in T47D breast cancer cells.

Knockdown of SF-1 and RNF31 affects components of steroidogenesis, TGFβ, and Wnt/β-catenin signaling in adrenocortical carcinoma cells.

MicroRNA-regulated gene networks during mammary cell differentiation are associated with breast cancer.

Interplay between AP-1 and estrogen receptor α in regulating gene expression and proliferation networks in breast cancer cells.

Estrogen receptors β1 and β2 have opposing roles in regulating proliferation and bone metastasis genes in the prostate cancer cell line PC3.

Estrogen receptor β expression induces changes in the microRNA pool in human colon cancer cells.

Studies in experimental autoimmune encephalomyelitis do not support developmental bisphenol a exposure as an environmental factor in increasing multiple sclerosis risk.

Liver × receptor ligands disrupt breast cancer cell proliferation through an E2F-mediated mechanism.

Support of a bi-faceted role of estrogen receptor β (ERβ) in ERα-positive breast cancer cells.

Oestrogen receptors in breast cancer: basic mechanisms and clinical implications.

Co-exposure to Phytoestrogens and Bisphenol A mimic estrogenic effects in an additive manner.