Recherche
Enseignement
Solutions
S'identifier
FR
EN - English
CN - 中文
DE - Deutsch
ES - Español
KR - 한국어
IT - Italiano
FR - Français
PT - Português
TR - Turkish
JA - Japanese
Division of Rheumatology,
Allergy and Clinical Immunology,
Division of Rheumatology, Allergy and Clinical Immunology
Iannis E. Adamopoulos has not added Biography.
If you are Iannis E. Adamopoulos and would like to personalize this page please email our Author Liaison for assistance.
Stimulation of osteoclast formation by inflammatory synovial fluid.
Virchows Archiv : an international journal of pathology Jul, 2006 | Pubmed ID: 16642388
In vitro biodegradation of three brushite calcium phosphate cements by a macrophage cell-line.
Biomaterials Sep, 2006 | Pubmed ID: 16720039
Macrophage-mediated biodegradation of poly(DL-lactide-co-glycolide) in vitro.
Journal of biomedical materials research. Part A Dec, 2006 | Pubmed ID: 16817218
Osteoclast differentiation and bone resorption in multicentric reticulohistiocytosis.
Human pathology Sep, 2006 | Pubmed ID: 16938523
Hepatocyte growth factor can substitute for M-CSF to support osteoclastogenesis.
Biochemical and biophysical research communications Nov, 2006 | Pubmed ID: 17022947
Immune regulation of bone loss by Th17 cells.
Arthritis research & therapy , 2008 | Pubmed ID: 18983698
Interleukin-17A upregulates receptor activator of NF-kappaB on osteoclast precursors.
Arthritis research & therapy , 2010 | Pubmed ID: 20167120
Myeloid DAP12-associating lectin (MDL)-1 regulates synovial inflammation and bone erosion associated with autoimmune arthritis.
The Journal of experimental medicine Mar, 2010 | Pubmed ID: 20212065
Structural, cellular, and molecular evaluation of bone erosion in experimental models of rheumatoid arthritis: assessment by μCT, histology, and serum biomarkers.
Autoimmunity Dec, 2010 | Pubmed ID: 20380588
Anti-IL-17A therapy protects against bone erosion in experimental models of rheumatoid arthritis.
Autoimmunity May, 2011 | Pubmed ID: 20925596
IL-23 is critical for induction of arthritis, osteoclast formation, and maintenance of bone mass.
Journal of immunology (Baltimore, Md. : 1950) Jul, 2011 | Pubmed ID: 21670317
Rheumatoid and pyrophosphate arthritis synovial fibroblasts induce osteoclastogenesis independently of RANKL, TNF and IL-6.
Journal of autoimmunity Dec, 2012 | Pubmed ID: 22867712
The emerging role of Interleukin 27 in inflammatory arthritis and bone destruction.
Cytokine & growth factor reviews Apr, 2013 | Pubmed ID: 23165310
The implication of vitamin D and autoimmunity: a comprehensive review.
Clinical reviews in allergy & immunology Oct, 2013 | Pubmed ID: 23359064
A mutation in mouse Pak1ip1 causes orofacial clefting while human PAK1IP1 maps to 6p24 translocation breaking points associated with orofacial clefting.
PloS one , 2013 | Pubmed ID: 23935987
NKG2C, HLA-E and their association with psoriasis.
Experimental dermatology Dec, 2013 | Pubmed ID: 24279916
The IL-23/IL-17 axis in psoriatic arthritis.
Autoimmunity reviews Apr-May, 2014 | Pubmed ID: 24424175
IL-17A gene transfer induces bone loss and epidermal hyperplasia associated with psoriatic arthritis.
Annals of the rheumatic diseases Feb, 2014 | Pubmed ID: 24567524
University of California, Davis
Shriners Hospitals for Children - Northern California
Dennis J. Wu1,
Neha Dixit1,
Erika Suzuki1,
Thanh Nguyen2,
Hyun Seock Shin1,
Jack Davis2,
Emanual Maverakis3,
Iannis E. Adamopoulos1,2
1Division of Rheumatology, Allergy and Clinical Immunology, University of California, Davis,
2Institute for Pediatric Regenerative Medicine, Shriners Hospitals for Children - Northern California,
3Department of Dermatology, University of California, Davis
Confidentialité
Conditions d'utilisation
Politiques
Contactez-nous
RECOMMANDER À LA BIBLIOTHÈQUE
NEWSLETTERS JoVE
JoVE Journal
Collections de méthodes
JoVE Encyclopedia of Experiments
Archives
JoVE Core
JoVE Business
JoVE Science Education
JoVE Lab Manual
Centre de ressources universitaires
Auteurs
Bibliothécaires
Accès
À PROPOS DE JoVE
Copyright © 2024 MyJoVE Corporation. Tous droits réservés.