Binu Tharakan

Botanical therapies in sexual dysfunction.

Antioxidant and DNA protecting properties of anti-fatigue herb Trichopus zeylanicus.

Botanical therapies in chronic fatigue.

Trichopus zeylanicus combats fatigue without amphetamine-mimetic activity.

Current concepts in apoptosis: the physiological suicide program revisited.

Apoptotic signaling induces hyperpermeability following hemorrhagic shock.

Neuroprotective mechanisms of ayurvedic antidementia botanical Bacopa monniera.

Anti-Parkinson botanical Mucuna pruriens prevents levodopa induced plasmid and genomic DNA damage.

Antiparkinson drug--Mucuna pruriens shows antioxidant and metal chelating activity.

Alpha lipoic acid attenuates microvascular endothelial cell hyperpermeability by inhibiting the intrinsic apoptotic signaling.

Angiopoietin-1 inhibits intrinsic apoptotic signaling and vascular hyperpermeability following hemorrhagic shock.

Mitochondrial complex III is involved in proapoptotic BAK-induced microvascular endothelial cell hyperpermeability.

Caspases - an update.

Alpha-lipoic acid attenuates hemorrhagic shock-induced apoptotic signaling and vascular hyperpermeability.

Centella asiatica extract selectively decreases amyloid beta levels in hippocampus of Alzheimer's disease animal model.

17beta-estradiol mediates protection against microvascular endothelial cell hyperpermeability.

Vascular leak in a rat model of preeclampsia.

Cyclosporine A prevents vascular hyperpermeability after hemorrhagic shock by inhibiting apoptotic signaling.

(-)-Deprenyl inhibits vascular hyperpermeability after hemorrhagic shock.

(-)-Deprenyl inhibits thermal injury-induced apoptotic signaling and hyperpermeability in microvascular endothelial cells.

17beta-estradiol mediated protection against vascular leak after hemorrhagic shock: role of estrogen receptors and apoptotic signaling.

Cyclosporine A--protection against microvascular hyperpermeability is calcineurin independent.

Curcumin inhibits reactive oxygen species formation and vascular hyperpermeability following haemorrhagic shock.

Inhibition of VE-cadherin proteasomal degradation attenuates microvascular hyperpermeability.

Role of β-catenin in regulating microvascular endothelial cell hyperpermeability.

β-Catenin dynamics in the regulation of microvascular endothelial cell hyperpermeability.

Differential effects of dopaminergic neurotoxins on DNA cleavage.

The effects of hypothermia and L-arginine on skeletal muscle function in ischemia-reperfusion injury.

Marrow stimulation improves meniscal healing at early endpoints in a rabbit meniscal injury model.

Inhibition of Fas-Fas ligand interaction attenuates microvascular hyperpermeability following hemorrhagic shock.

Microvascular endothelial cell hyperpermeability induced by endogenous caspase 3 activator staurosporine.

Tumor necrosis factor-related apoptosis-inducing ligand promotes microvascular endothelial cell hyperpermeability through phosphatidylinositol 3-kinase pathway.

Regulation of tumor necrosis factor-α-induced microvascular endothelial cell hyperpermeability by recombinant B-cell lymphoma-extra large.

The effects of inflammatory cytokines on lymphatic endothelial barrier function.

Doxycycline attenuates burn-induced microvascular hyperpermeability.

Reactive oxygen species-caspase-3 relationship in mediating blood-brain barrier endothelial cell hyperpermeability following oxygen-glucose deprivation and reoxygenation.

Vascular hyperpermeability and aging.

Melatonin inhibits thermal injury-induced hyperpermeability in microvascular endothelial cells.

The role of intrinsic apoptotic signaling in hemorrhagic shock-induced microvascular endothelial cell barrier dysfunction.

Tumor necrosis factor-α disruption of brain endothelial cell barrier is mediated through matrix metalloproteinase-9.

Tumor necrosis factor-α-induced microvascular endothelial cell hyperpermeability: role of intrinsic apoptotic signaling.

Blood-brain barrier dysfunction following traumatic brain injury.